yes, that's right - big randomised controlled trials that showed if you decrease your saturated fats, you lower your risk of heart disease. If you go to Google Scholar you can see pages and pages of academic articles about it:http://scholar.google.co.uk/scholar?hl= ... 5&as_sdtp=
Ummmm, no they didn't: almost every high-quality (prospective) observational study ever conducted has found that saturated fat intake is not associated with heart attack risk (or with increased cholesterol):http://www.ncbi.nlm.nih.gov/pubmed/20071648http://www.ajcn.org/content/77/5/1146.shorthttp://www.ncbi.nlm.nih.gov/pubmed/1386252http://www.ajcn.org/content/67/5/828.shorthttp://www.sciencedirect.com/science/ar ... 0711003145
A meta-analysis of prospective epidemiologic studies showed that there is no significant evidence for concluding that dietary saturated fat is associated with an increased risk of CHD or CVD. More data are needed to elucidate whether CVD risks are likely to be influenced by the specific nutrients used to replace saturated fat.
Saturated fat is only implicated in heart disease as part of the defunct: "diet-heart hypothesis", which states that increased cholesterol causes heart disease, saturated fat increases cholesterol, therefore saturated fat causes heart disease.
This is wrong because (a) it's based on childish logic and (b) saturated fat has only be observed to increase cholesterol in short term trials, over the longer term it has no significant effect.
Stephan Guyenet has a great overview of all that research that links saturated fat consumption with increased cholesterol (spoiler alert: there is none) http://wholehealthsource.blogspot.com/2 ... rease.html
Don't take my word for it (or even Stephan's), you can listen to one of the leading researchers in the field telling you that carbohydrates increase cholesterol, not saturated fats:http://www.meandmydiabetes.com/2012/04/ ... t-depends/
We looked carefully at the saturated fat effects. With more saturated fat in the diet, we did see a signal for an increase in the overall amount of cholesterol in their blood. But when we looked more carefully, that slightly increased amount of total cholesterol was not being carried by more of the dangerous, small particle LDLs. It seemed to be carried more by larger particles. Actually, in the people eating more fat, and fewer carbs, the total particle concentration, which most people in our field think is a stronger signal of risk that total cholesterol, the total number of particles did not go up.
When people ate more fat and less carbohydrate, the number of small particle LDLs remained low, and switching from monounsaturated to saturated fat didn’t increase their number at all. In fact, when people switched from mono- to saturated fat in this study, the large particle LDLs might have gone up a little bit, and the small particles went down. So by anybody’s current criteria about whats’s important for heart disease risk, saturated fat caused no increase in risk. That was clear to people who understand the role to the lipoprotein particles, as opposed to the overall cholesterol level, which I’m sure for some people is a subtle distinction.