The Global Diabetes Community Forum
http://www.sciencedaily.com/releases/20 ... 134833.htm
"metabolites can also be used as biomarkers to precisely determine the risk of diabetes at a very early stage"
Our findings, which highlight five branched chain and aromatic amino acids from 61 metabolites profiled, are particularly noteworthy in the context of experimental and clinical data suggesting that certain amino acids may be both markers and effectors of insulin resistance
In addition to insulin resistance, impaired insulin secretion plays a critical role in the pathogenesis of type 2 diabetes. In this regard, it is noteworthy that multiple amino acids, particularly the branched chain amino acids, are modulators of insulin secretion24-26. Thus, another possible mechanism by which hyperaminoacidemia could promote diabetes is via hyperinsulinemia leading to pancreatic α-cell exhaustion.
hanadr wrote:Having merely skimmed all this data. Thanks for posting it!!!
I think it supports the theory that T2 is a genetic metabolic error of which weight gain is a symptom not a cause and patients are NOT responsible for causing their own condition.
They also found no significant difference between the screened and non-screened groups in the number of deaths specifically attributable to diabetes, cardiovascular illness, cancer, or other causes of death.
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