Let's just make sure we are clear here though.Triglycerides are one of the ways that fat is transported around the human body for use as energy. They are also stored in the liver (rather like glycogen is for glucose) and when the body requires free fatty acids as an energy source, glucagon triggers the liver to undertake the conversion.
In the context of NAFL, the liver is holding too much triglyceride in the hope that it is needed as an energy source.
The storage of fat on the form of trigs requires special cells called adipocytes. In the case of excess fat needing storage, the body simply builds and fills more adipocytes, and there is no pre designed upper limit on this process. BUT once an adipocyte is created from a spare stem cell then it cannot be reversed, and this is why adipose tissue is so difficult to shift, Ketosis only empties the cell contents, and refills them when more fat becomes available. So our spare tyres inflate, deflate, but NEVER disappear. Cellulite is a form of adipose tissue, and can only be removed by vacuming. Not so for the glucose stores which can be any cell in the body apart from brain cells or nerve cells. The normal place is muscle tissue, which can be deleted by fasting.
The body can store about 100g of glycogen in the liver, and 500g in the muscles, but then the store becones full, and excess is secreted via the kidneys. Excess fat creates its own elbow room as required.
Adipose tissue congregates around the vital organs (heart, liver, kidneys, stomach, gut) and so is called visceral fat since it is protecting the vitals. Adipose tissue also exists in other areas of the body as subcutaneous fat, and on the back as brown fat that acts as a built in heater. These secondary clusters tend to naturally deplete with age, but not by diet or exercise.
So as I get older, my hair migrates downward, and my adipose tissue migrates to my midriff, and I feel the cold more keenly. Ketosis may reduce my beergut temporarily, but the flab remains.
An interesting article on Fructose is available:
http://www.medscape.com/viewarticle/777692