Interesting hypothesis Loobles; especially when we consider the anorexic body serially closes down un-used or under-used functions in its' quest to survive. I can buy that.
Binge eating disorder and Bulimia are associated with the development of T2 (and eating disorders are also associated with people who have bothT1 and T2 diabetes; the focus on diet being one of the factors )
This paper suggests why people with Bulimia ( bingeing/purging) have a higher risk of later developing T2 .
https://escholarship.org/uc/item/6vt2k42t( ie hyperinsulinemea / high levels of insulin that are released during the bingeing periods)
However, it also mentions that one trial showed that three years after therapy, those that had managed to overcome their disorder had a lower glucose level than those who still had an active eating disorder .
For Anorexia where there is a definite calorie deficit, there isn't really that much evidence about a link with later T2.
Most studies say that people with active anorexia are insulin sensitive (a few papers the opposite though).
Diabetes is not usually given as one of the long term effects .
However, even a small increase in fat when in recovery ,tends to be on the trunk. This has been shown to be accompanied by an increase in insulin resistance . (this very obvious increase in fat around the middle can sadly trigger a return to anorexia )
http://onlinelibrary.wiley.com/doi/10.1111/j.1365-2265.2011.04046.x/full
To me that makes sense because if the fat cells in the lower body which is the normal safe haven for fat have shrunk then it's likely that fat is going to be deposited where it shouldn't be.
There is some evidence that fat distribution seems to normalise after complete, long term weight restoration (but the authors point out that evidence is very scarce)
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4179194/