Some people produce normal amounts of insulin but for various reasons the cells that need it to act on the glucose we have eaten are rejecting some or most of it, so the glucose floats about in our blood stream with nowhere to go. This is insulin resistance, put very simply.
Other people do not produce enough insulin (or none at all) so again, the cells that need it to convert the glucose in to energy are not getting enough, or none at all. Again, the glucose floats about in the bloodstream. They may or may not have insulin resistance.
Brilliant explanation, maybe you should get a job writing the how to manuals for some of the big companies who give out pages of gobbledegook instead of plainly written instructions.
The problem with that is the gobbledygook may be the correct version.
Some people produce normal amounts of insulin but for various reasons the cells that need it to act on the glucose we have eaten are rejecting some or most of it, so the glucose floats about in our blood stream with nowhere to go. This is insulin resistance, put very simply.
Other people do not produce enough insulin (or none at all) so again, the cells that need it to convert the glucose in to energy are not getting enough, or none at all. Again, the glucose floats about in the bloodstream. They may or may not have insulin resistance.
So how do you know if you've got both, seeing as the end result is the same?
So diet is still essential as far as treating both scenarios ?
Insulin resistance is, in part, brought on by being overweight, and particularly by having too much fat round the liver and pancreas both inside and outside the organs. If a type 2 is overweight, especially round the waist line, and struggles to keep fasting levels down when levels later in the day are going down well, it is likely due to insulin resistance. ........
........Insulin resistance can also cause weight gain in the first place because the insulin produced isn't getting used, so like the glucose, it floats around in the bloodstream but is then stored as fat. Vicious circle sometimes! IR can be improved by weight loss.
So high morning levels and liver dumps are probably because of insulin resistance?
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Hi. If you have been losing weight without really trying then late onset T1 becomes a possibility. This is when the pancreas islet cells start to fail and there isn't enough insulin to use up blood glucose and the body turns to burning it's stored fat to get energy hence the weight loss. This can happen either thru antibody or viral damage to the pancreas or cell death due to excessive insulin production thru long-term insulin resistance. If you continue to lose weight to a level below a normal BMI then ask for tests for Late onset T1 (LADA). You will, of course lose weight thru a low-carb diet but LADA tends to occur as a surprise and despite increasing carbs to try to compensate for excessive weight loss. Complicated isn't it!So high morning levels and liver dumps are probably because of insulin resistance?
If that is the case (and I think I might have been insulin resistant long before I have diabetes) it's strange that I've been losing weight without even trying over the last year or so. I have to have 3-monthly hospital check-ups and I've lost around half a stone each time. I still have around 2 stone to lose.
Hi. If you have been losing weight without really trying then late onset T1 becomes a possibility. This is when the pancreas islet cells start to fail and there isn't enough insulin to use up blood glucose and the body turns to burning it's stored fat to get energy hence the weight loss. This can happen either thru antibody or viral damage to the pancreas or cell death due to excessive insulin production thru long-term insulin resistance. If you continue to lose weight to a level below a normal BMI then ask for tests for Late onset T1 (LADA). You will, of course lose weight thru a low-carb diet but LADA tends to occur as a surprise and despite increasing carbs to try to compensate for excessive weight loss. Complicated isn't it!