I think (and this is based on no more than hanging around the forum, and reading both the T1 and the T2 threads), is that there is a vast difference between the two. It comes down to beta cells and insulin resistance.
T1s have already lost (or are rapidly losing) their beta cells. Its auto immune. They are dying. T1s have lots of valid concerns about maintaining bg at their target levels, to prevent all the complications, but sooner or later their beta cells are gonna kick the bucket, or they have already done so. Their complications are going t9 be mainly from high bgs damaging nerves and blood vessels, and so on.
On the other hand, T2s are killing healthy functioning beta cells every time their blood glucose rises high enough. That is generally considered to be above 7.8 mmol/l ( yes, there is some debate on that, but whatever the actual figure is, damage happens above that figure). So for a T2, every time you go above 7.8mmol/l you are slowly and incrementally killing more of the beta cells, raising insulin resistance, raising the knock on risks of insulin resistance (heart disease, Alzheimers, strokes, etc.), AND risking the same damage that T1s get with high blood glucose. T2s are also, spike by spike, inching closer to more medication and eventual insulin as their beta cells die off.
So I totally get why T2s are often preoccupied with not going above the 7.8mmol/l ceiling.
(Of course, T1s with insulin resistance get the disadvantages of both worlds, minus the beta cells.)