@Melgar - the pain you are describing (radiating up into the back) - is spookily similar to what my wife struggled with before having a major episode, and having her gall bladder removed.
(I may be thinking the same thing as
@Antje77 - maybe not).
At the time, we were confused - how could she have a gall stone? she hardly eats fat.. but it seems that not eating fat means that the gall bladder is not regularly flushed out - it's a storage for bile to break down fat after all - so it isn't people who eat too much fat who get gallstones, it's people who don't eat enough.
Usual caveats - not making a diagnosis -
Thanks for your clarification on shelf life, and sure... it was badly put on my side, I was just chewing away on the thought that internally produced insulin doesn't have a shelf life.. and went from there... (incidentally, I don't disagree; but I think C-Peptide is a more reliable marker of internally produced insulin, because there is no equivalent in artificial insulin, regardless of shelf life out of the body)
.. and absolutely I cannot know what's going on in your case. You know I'm not making light of it, and I think I'm being helpful. I`ve no doubt you'll put me right if not (more for anyone else reading).
Clearly you have hypos, so you are producing insulin. granted. Let me try to lay out what I was thinking - Ketoacidosis is dis-regulated ketone production. The liver has access to glucose and free fatty acids. Because there no insulin to "tell" it to do anything with the glucose, the liver churns out ketones as a survival mechanism, but there is nothing to "tell" the liver to "stop, that's enough".. because, no insulin.
With your diet.. the blood glucose variation you see can only be on account of what your liver is doing.. so how does one account for the lows (well, that's easy, you consume all the available energy because you are so active) as well as the highs (well, that's more difficult - but again, where else can it come from other than the liver)... so is there any way that the liver can carry on producing glucose in a dis-regulated way like it does in ketoactidocis.. well, potentially, if you are not having the balancing effect of fat.
Once you use up the available glycogen, you should tip into fat burning (that's the entire point of the fasting I do). But, you will get to that point much quicker with the exercise you do. what happens if there is no fat to burn? well, you will have lots of Glucagon "telling" your liver to do all the things it does.. including generating glucose from whatever it can.
The resulting glucose flows out to the rest of your body - it doesn't go to your pancreas, so you don't get the insulin response (at least not immediately) - so you are in a kind of dis-regulated state of glucose production...
it's just a theory, but it's the best I've got for the moment.