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<blockquote data-quote="AloeSvea" data-source="post: 989243" data-attributes="member: 150927"><p>I was very excited to find this thread, as I have a very similar profile to seadragon and donnadoobie - complete with the same high cholesterol donnadoobie has, and frustration with FBG, and not quite being able to get to the 30s HBA1c/'normal BG levels. (And BakedAlaska's understanding of the positive role alcohol can have for relaxing of an evening and lowering/ the next morning's BG!)</p><p></p><p>Since exercising at a much higher level with H.I.T.-sprints, and regular bench-pressing and/or ab exercises, at the same time as low-carbing, I have seen my FBG rise to mid 6s, from an initial happy FBG 5-point-something target for a couple of weeks when I first started seriously low carbing in July. (I had been 50-100-150g carbing before that I think - not sure as I wasn't counting, but probably depending if I was eating my beloved sweet potatoes/kuumara.) (Sigh.) Anyway. Now the prediabetic level of the low to higher 6s, with some drops to healthier BG during the day post-meal (bizarrely! but not for folk with Dawn Phenomenon/liver dumping issues I suppose.)</p><p></p><p>I feel like I have an intimate, ongoing and unhappy relationship with the mechanisim/s in our livers that no longer responds to insulin (with insulin receptors that aren't there or not working?) or there isn't enough insulin,although personally I don't think that not enough insulin is the situation for diabetics such as us - as we CAN get our BGs down to healthier levels - if only our liver would play non-diabetic ball! Which is our cross to bear? I live in hope this improves with time. </p><p></p><p>The diabetic cross we are bearing seems to be the consequence of our livers responding to even small fatty liver levels (which is Prof Taylor's theory of T2D development also) mucking everything up. It's complicated! I can't really get my head around it at one time all the mechanisms are so complicated. I have found these three mechanisms at least that contribute to diabetic DP/leaky livers/liver dumping diabetics. (Don't ask me to talk about these over dinner! I won't remember them! ;-).) But the genetic disposition to T2D that is talked about is more than likely a genetic disposition to being very sensitive to fatty liver, for diabetics like us at any rate. The ol' fat around the middle thing. (At the same time as being quite physical or even athletic - as we are? I would say so.) </p><p></p><p>The role of Acrp30: <a href="http://www.ncbi.nlm.nih.gov/pmc/articles/PMC209474/" target="_blank">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC209474/</a></p><p>The role of the FOX6: <a href="http://www.diabetesselfmanagement.com/blog/healing-leaky-livers/" target="_blank">http://www.diabetesselfmanagement.com/blog/healing-leaky-livers/</a></p><p>The role of CRTC2: <a href="http://consumer.healthday.com/circulatory-system-information-7/blood-disorder-news-68/constant-high-blood-sugar-disables-insulin-off-switch-613284.html" target="_blank">http://consumer.healthday.com/circulatory-system-information-7/blood-disorder-news-68/constant-high-blood-sugar-disables-insulin-off-switch-613284.html</a></p><p></p><p>Why switching to burning fat - ketogenisis - helps: <a href="http://dtc.ucsf.edu/types-of-diabetes/type1/understanding-type-1-diabetes/how-the-body-processes-sugar/the-liver-blood-sugar/" target="_blank">http://dtc.ucsf.edu/types-of-diabetes/type1/understanding-type-1-diabetes/how-the-body-processes-sugar/the-liver-blood-sugar/</a></p><p></p><p>Which is why I think we need to 'stand by' and give our bodies time? if they/our livers are going to heal in this way. If they can! Another way of thinking about it, which is how I see it, is that it took me three decades for my liver to so powerfully begin to malfunction (I had signs of insulin resistance and metabolic syndrome from my mid 20s, but be ******** if I ever heard either of those phrases or understood that back in the day!). (I doubt that my GP did either! In fact I know she didn't or she would have mentioned it. She DID keep checking my BG levels though, as she obvilously knew the risk for T2D. But I wasn't seeing her in the two years when my body tipped over into diabetes.) If I can get normal functioning of my liver again, maybe it is going to take a while?</p><p></p><p>And living in hope isn't a bad thing?</p><p></p><p>I am waiting to see if I can deplete my liver of the fat, if that is indeed the issue, over time, will help my liver regain proper function of that awfully complex blood glucose regulating system. I am doing it with I.F.ing and periodic fasting - gently! Or as gently as possible. Over time. And then - as many of us diabetics trying out various things - to wait and see.</p><p></p><p>And now I can follow you guys in your getting as-healthy-as-possible journey too! As you struggle with the same diabetic issues as I am...</p></blockquote><p></p>
[QUOTE="AloeSvea, post: 989243, member: 150927"] I was very excited to find this thread, as I have a very similar profile to seadragon and donnadoobie - complete with the same high cholesterol donnadoobie has, and frustration with FBG, and not quite being able to get to the 30s HBA1c/'normal BG levels. (And BakedAlaska's understanding of the positive role alcohol can have for relaxing of an evening and lowering/ the next morning's BG!) Since exercising at a much higher level with H.I.T.-sprints, and regular bench-pressing and/or ab exercises, at the same time as low-carbing, I have seen my FBG rise to mid 6s, from an initial happy FBG 5-point-something target for a couple of weeks when I first started seriously low carbing in July. (I had been 50-100-150g carbing before that I think - not sure as I wasn't counting, but probably depending if I was eating my beloved sweet potatoes/kuumara.) (Sigh.) Anyway. Now the prediabetic level of the low to higher 6s, with some drops to healthier BG during the day post-meal (bizarrely! but not for folk with Dawn Phenomenon/liver dumping issues I suppose.) I feel like I have an intimate, ongoing and unhappy relationship with the mechanisim/s in our livers that no longer responds to insulin (with insulin receptors that aren't there or not working?) or there isn't enough insulin,although personally I don't think that not enough insulin is the situation for diabetics such as us - as we CAN get our BGs down to healthier levels - if only our liver would play non-diabetic ball! Which is our cross to bear? I live in hope this improves with time. The diabetic cross we are bearing seems to be the consequence of our livers responding to even small fatty liver levels (which is Prof Taylor's theory of T2D development also) mucking everything up. It's complicated! I can't really get my head around it at one time all the mechanisms are so complicated. I have found these three mechanisms at least that contribute to diabetic DP/leaky livers/liver dumping diabetics. (Don't ask me to talk about these over dinner! I won't remember them! ;-).) But the genetic disposition to T2D that is talked about is more than likely a genetic disposition to being very sensitive to fatty liver, for diabetics like us at any rate. The ol' fat around the middle thing. (At the same time as being quite physical or even athletic - as we are? I would say so.) The role of Acrp30: [URL]http://www.ncbi.nlm.nih.gov/pmc/articles/PMC209474/[/URL] The role of the FOX6: [URL]http://www.diabetesselfmanagement.com/blog/healing-leaky-livers/[/URL] The role of CRTC2: [URL]http://consumer.healthday.com/circulatory-system-information-7/blood-disorder-news-68/constant-high-blood-sugar-disables-insulin-off-switch-613284.html[/URL] Why switching to burning fat - ketogenisis - helps: [URL]http://dtc.ucsf.edu/types-of-diabetes/type1/understanding-type-1-diabetes/how-the-body-processes-sugar/the-liver-blood-sugar/[/URL] Which is why I think we need to 'stand by' and give our bodies time? if they/our livers are going to heal in this way. If they can! Another way of thinking about it, which is how I see it, is that it took me three decades for my liver to so powerfully begin to malfunction (I had signs of insulin resistance and metabolic syndrome from my mid 20s, but be ******** if I ever heard either of those phrases or understood that back in the day!). (I doubt that my GP did either! In fact I know she didn't or she would have mentioned it. She DID keep checking my BG levels though, as she obvilously knew the risk for T2D. But I wasn't seeing her in the two years when my body tipped over into diabetes.) If I can get normal functioning of my liver again, maybe it is going to take a while? And living in hope isn't a bad thing? I am waiting to see if I can deplete my liver of the fat, if that is indeed the issue, over time, will help my liver regain proper function of that awfully complex blood glucose regulating system. I am doing it with I.F.ing and periodic fasting - gently! Or as gently as possible. Over time. And then - as many of us diabetics trying out various things - to wait and see. And now I can follow you guys in your getting as-healthy-as-possible journey too! As you struggle with the same diabetic issues as I am... [/QUOTE]
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