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Reactive Hypoglycemia
New Paradigm For Insulin Resistance
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<blockquote data-quote="LittleGreyCat" data-source="post: 1235204" data-attributes="member: 6467"><p>One further thought; if in fact T2 diabetes is all about the efficiency of fat cells, and the effects of over loading them, then this might explain other things.</p><p></p><p>Roy Taylor's theory about everyone having a "fat threshold" might be down to the ability of fat cells to process glucose and running out of spare capacity.</p><p></p><p>This would explain how people who are obese can go on a crash diet (which RT has confirmed targets a fatty liver) and then reverse T2 symptoms. A percentage loss of weight can reverse T2 symptoms whilst the person is still carrying a lot of fat. Perhaps this is because all those large and extra fat cells provide much more overall processing capacity for glucose even though the individual cells are impaired.</p><p></p><p>This in turn would tie in with skinnier people still having T2 problems; they perhaps have fewer active fat cells and so less overall processing capacity. In turn they have to at least partially deplete most of their fat cells (and lose more weight even though apparently of "normal weight") until the processing capacity of their fat cells is sufficient to manage the BG.</p><p></p><p>This in turn might mean that if you are carrying a lot of active fat cells, you are more easily able to manage your BG levels by partially depleting them to match the spare capacity to your glucose intake.</p><p></p><p>New strategy; put on two stone then take off one?</p><p>I wish.<img src="data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7" class="smilie smilie--sprite smilie--sprite8" alt=":D" title="Big Grin :D" loading="lazy" data-shortname=":D" /></p></blockquote><p></p>
[QUOTE="LittleGreyCat, post: 1235204, member: 6467"] One further thought; if in fact T2 diabetes is all about the efficiency of fat cells, and the effects of over loading them, then this might explain other things. Roy Taylor's theory about everyone having a "fat threshold" might be down to the ability of fat cells to process glucose and running out of spare capacity. This would explain how people who are obese can go on a crash diet (which RT has confirmed targets a fatty liver) and then reverse T2 symptoms. A percentage loss of weight can reverse T2 symptoms whilst the person is still carrying a lot of fat. Perhaps this is because all those large and extra fat cells provide much more overall processing capacity for glucose even though the individual cells are impaired. This in turn would tie in with skinnier people still having T2 problems; they perhaps have fewer active fat cells and so less overall processing capacity. In turn they have to at least partially deplete most of their fat cells (and lose more weight even though apparently of "normal weight") until the processing capacity of their fat cells is sufficient to manage the BG. This in turn might mean that if you are carrying a lot of active fat cells, you are more easily able to manage your BG levels by partially depleting them to match the spare capacity to your glucose intake. New strategy; put on two stone then take off one? I wish.:D [/QUOTE]
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