Hi, my name's Andrew, I’m 29 from Leeds. I was diagnosed on my birthday this April but I wasn't too shocked by the diagnosis as my dad has T1 and his dad had T1. I did however associate T2 with being overweight and as all my life I have kept my BMI below 25, I was shocked a little. Now however I am just shocked by my naivety and lack of knowledge more than anything else.
At diagnosis my HBA1C was 10.1 so I was put on metformin but due to some bad side effects I was only on it for 1 day them my brilliant GP said to try just diet and exercise first. I can now happily say that after 3 months of exercising more and reducing my carbs I have got my HBA1C down to 6.8. Still a bit to go but hey, I’m happy with my effort so far.
The reason for my post is I don't seem to come across many T2's who were diagnosed under the age of 30 which kind of worries me for the future because when I was diagnosed, I was told (not by a specialist) that T2 suffers would last approx 30 years from diagnosis. I don't really want to die before I’m 60!!!
Looking forward to getting to know you all and really hope the 30 year thing is a load of rubbish!
Yeah, me and my twin were diagnosed when we were both 12. He was diagnosed before me though :wink:
Since then, we were lab rats for diabetes research. Since we have identical DNA, they could see the slight differences when he had been diagnosed and I hadn't.
At the recent DUK event in Birmingham, I met a young girl, who has been T2 for several years. she was a slender creature, but assured me she hadn't always been. She was a University student, so presumably about 20
If T2 is linked to allergic responses as has recently been suggested, I expect we'll find that quite a few youngsters, who have been assumed to be T1, because of their ages, are actually T2s.
I'll fish about for information
I came across this in a paper about insulin resistance in the offspring of T2 diabetics. the hypothesis that insulin resistance in the skeletal muscle of insulin-resistant offspring of patients with type 2 diabetes is associated with dysregulation of intramyocellular fatty acid metabolism, possibly because of an inherited defect in mitochondrial oxidative phosphorylation.
I already knew that T2 is strongly genetic.