Hi
@lindijanice, I agree with you.
Let us not be too precious about reporting one's BSLs nearer the lower end of the normal range ( and the number of hypos which occur as a result) and bemoaning the presence of BSLs near the upper end.
I try to answer posts about why a person's BSL is definitely higher or lower that the expected range but reporting BSL as a routine rather than for sharing experience or asking a question does seem rather trite in some ways (putting on helmet, kevlar vest and gas mask as I type)!!
All of which begs the question of why does a human, non-diabetic body's physiology usually maintain BSLs in the stated range?
Our brain and emergency bodily responses certainly tend to let us know when BSLs are too low. Although the fact that our brain might use ketones as fuel in certain circumstances might muddy this a bit. But still, too low is disastrous for the brain.
We know that sustained BSLs above a certain level cause harm long term. Too much glucose hanging around for too long leads to things called AGEs (Advanced Glycation Endproducts) which are thought to contribute to the many of the complications of diabetes. see clinical.diabetesjournals.org Glucose, AGEs and Diabetes Complications Clinical Diabetes Oct 2003 Peppa et al
We might assume that upper limit of normal BSLs both fasting and after food in a non-diabetic is a compromise.
On one hand having sufficient glucose in the blood after a meal to then enter the body cells whether with insulin's aid or without (e.g. the latter into brain and liver cells) and use of liver storage of glucose, released between meals, to keep BSLs from dropping too low plus the liver's ability to convert some ingested protein to glucose and on the other hand having some glucose sticking to proteins in the body to form AGEs.
Maybe some level of AGEs can be tolerated because the body has ways to protect itself against AGEs up to a certain point and the above article points out that treatment by the food industry has increased pre-AGE products in food. So excessive glucose in the blood is not the only source of AGEs that we as diabetics need to worry about.
We also know that if our body has to secrete more and more insulin or we inject more and more insulin as diabetics that excessive insulin has its downsides too. see diabesity.ejournals.ca Hyperinsulinaemia: A unifying theory of chronic disease? Diabesity 2015 1 (4) Crofts et al.
And we have the study about cardiovascular disease and diabetes; see" HBA1C as a risk factor for cardiovascular outcomes and all-cause mortality in diabetic and non-diabetic populations: a systemic review and meta-analysis" Cavero-Redondo et al BMJ Open Vol.7 issue & 2015 where the least mortality was found to be with HBA1C readings between 6.0 and 8.0 % for diabetic populations and between 5.0 and 6.0 % for non-diabetic populations. So is vying for an HBA1C less than 6% (42mmol/mol) wise?
Of course we do not know why the death rate may increase when a diabetic maintains a HBA1C less then 6% (42mmol/mol) ? more hypos and how predictive are statistics and how accurate and reliable is HBA1c as a marker of diabetes control when it measures an average not the range of BSLs over time.
As diabetics we do the best we can. I admit that no one can stop people posting their results but is it really necessary?
Do you seek a weather report that tells you and others that the wind speed is precisely 4 knots from the NNW when you know that is just in one place and that it will very likely to different everywhere else OR is saying the windspeed is within the predicted range of 3 to 7 knots from the NNW to NNE good enough?