Useless Pretty Boy said:Spiral said:I'd like to ask why type 2's are being prescribed insulin in the first place. Whatever happened to diet control? And no, I'm not advocating the extreme ow carb diet, just eating healthily... as if it were difficult. Sigh.
This kind of smug **** shows no understanding of T2 diabetes.
My weight is a symptom of diabetes not the cause. I have genes which are better suited to a feast/famine lifestyle. This might have been a huge evolutionary advantage in times past, but unfortunately, late 20th early 21st century life is a permanent feast and my T2 body is very efficient at laying the excess down as fat.
The fat adds to insulin resistance, which means that my body does not use the insulin it does produce properly. T2s ironically start off by overproducing insulin. Those large quantities of insulin mean that all that circulating glucose (from a diet high in carbohydrate) gets laid down as fat.
Carbs are also very addictive and those extreme blood sugar swings feed carb crafings. I was permanently hungry when I was eating the Healthy Plate, always on the lookout for my next mouthful, in spite of having eaten a meal high on starchy carb which was supposed to fill me up. It is only since I stopped eating them that I have been able to identify the difference between carb cravings and real hunger.
Those toxically high sugar levels continue to damage virtually every system in the body, including the beta cells in the pancreas which produce the insulin in the first place, eventually killing them off. So diabetics who are prescribed insulin usually have very poor insulin production, I'd have thought you knew that UPB.
Yeah, but there simply isn't any way for me to feel sorry for anyone who has eaten themselves into diabetes. I'm sure a lot of people with T2 would rather look at their weight as a symptom than a cause, because then it isn't their fault.
Simply not being able to stop eating isn't a medical condition, no more than feeling sad every now and then is. But instead of just living with the sadness every now and then, we're becoming a country of pill poppers. And instead of simply not gorging, we start prescribing expensive insulin instead. I'm a type 1. I'll die within a month if unable to inject insulin. Slowly and painfully. Type 2's won't. So I just can't bring myself to feel any sort of sympathy. I'd give anything to be type 2 instead of type 1, but instead I'm stuck with my needles (and I'd like to point out I've had to get past my needle phobia in order to inject) while type 2's blame their genetics for not being able to stop eating.
That just doesn't wash with me.
but the fact remains that we are all in charge of what we put into our mouths. Nobody suddenly becomes obese, and a set of ordinary scales are not expensive.
http://www.phlaunt.com/diabetes/14046739.phpEven so, it is likely that you've been told that you caused your diabetes by letting yourself get fat and that this toxic myth is damaging your health.
Blaming you for your condition causes guilt and hopelessness. Even worse, the belief that people with diabetes have brought their disease on themselves inclines doctors to assume that since you did nothing to prevent your disease, you won't make the effort to control it--a belief that may lead to your getting extremely poor care.
The myth that diabetes is caused by overeating also hurts the one out of five people who are not overweight when they contract Type 2 Diabetes. Because doctors only think "Diabetes" when they see a patient who fits the stereotype--the grossly obese inactive patient--they often neglect to check people of normal weight for blood sugar disorders even when they show up with classic symptoms of high blood sugar such as recurrent urinary tract infections or neuropathy.
Adiponectin is a protein hormone that modulates a number of metabolic processes, including glucose regulation and fatty acid catabolism.[3] Adiponectin is exclusively secreted from adipose tissue into the bloodstream and is very abundant in plasma relative to many hormones. Levels of the hormone are inversely correlated with body fat percentage in adults,[4] while the association in infants and young children is more unclear. The hormone plays a role in the suppression of the metabolic derangements that may result in type 2 diabetes,[4] obesity, atherosclerosis,[3] non-alcoholic fatty liver disease (NAFLD) and an independent risk factor for metabolic syndrome.[5]
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