Hi Alison Campbell. I checked the paste and the estimate of 5g of carbs won't be far out for the quantity I ate.Sorry, I appreciate it can be difficult testing around work breaks but I don't think you can make a judgement about what is high at 40 mins when you did not test before the meal.
If you want us to check the paste for you just ask by telling us the brand details and approx portion used.
I believe Jim is giving a fair representation of the book.
A “decade’s worth” isn’t literal glucose but overall impact including the pancreas and the sensitivity that will take time to recover.
This doctor is getting results.
So can insulin resistance be reversed. I thought it could only be managed.I believe Jim is giving a fair representation of the book.
A “decade’s worth” isn’t literal glucose but overall impact including the pancreas and the sensitivity that will take time to recover.
This doctor is getting results.
Hi Alison Campbell. I checked the paste and the estimate of 5g of carbs won't be far out for the quantity I ate.Sorry, I appreciate it can be difficult testing around work breaks but I don't think you can make a judgement about what is high at 40 mins when you did not test before the meal.
If you want us to check the paste for you just ask by telling us the brand details and approx portion used.
There a two things springing to mind here Jim. Firstly despite usually eating LCHF quite strictly my weight loss has been satisfactory but I've never seen even a trace of ketones when using a pee stick. Perhaps my body still has access to too much glucose regardless of what I eat.
Secondly perhaps I need to up my efforts to fast to reduce the stored glucose quicker.
So can insulin resistance be reversed. I thought it could only be managed.
I could be misinterpreting parts of the book. As I remember it, once glycogen stores are full and the fat cells throughout the body are fully engorged, if glucose continues to enter the system, then it has nowhere to go other than blood, organs and tissues. Hypoglycaemics and exogenous insulin then force it out of the blood and further into those tissues. They don’t dissolve the glucose, they simply move it somewhere else. According to Fung, this is reflected in data that shows that these medications, despite lowering blood glucose, don’t by themselves improve complication outcomes. Whether or don’t this is true I can’t say.
This is certainly how it’s all explained in the book, but for sure he could be simplifying for the audience, and I could have misunderstood some finer details. In any case, it’s a fascinating read for sure. Might give it a third listen
However, if insulin becomes excessive, then the body is always trying to store glycogen and fat. Since there’s not much room for glycogen, it produces fat. (Note – this is normal. This process reverses during fasting) The liver exports this fat out as triglycerides along with very low density lipoprotein (VLDL) to other organs but particularly to fat cells called adipocytes.
Now, adipocytes are specialized cells to store fat. Having more fat cells is not particularly dangerous. That’s what it does. Other than taking up room, it doesn’t really matter. The fat cell is designed to hold fat, so you don’t get sick from from it. Obesity itself is not the cause of the problem. The critical problem occurs when you get fat where it’s not supposed to be.
Hi everyone
I'm still struggling a bit to understand what sometimes causes raised BG and today has been typical.
I've actually given up testing first thing in the morning as it always seems to be high with liver dump and it's quite frustrating but I still test after meals (2 hours usually). Today due to work I only got chance to test for the first time 40 minutes after lunch. I then went for a 20 walk, tested again, then after the usual 2 hours.
The first test was a bit of a 1 off. I don't usually test that soon after eating. The reading was 9.6 mmol/l which I thought was high for what I had eaten. After a walk I tested again and as expected it had dropped, 6.7 mmol/l. But when I tested at the usual 2 hours it had gone up to 8 mmol/l. What's that all about?
I had scrambled eggs and 3 rashes of bacon for breakfast and lunch was a thai curry that I reckon had 5g of carbs in the curry paste plus whatever was in the chicken thighs, onion, coconut cream and coconut oil.
The only thing I'd had after lunch was a black coffee and a glass of water.
Doesn't that seem high for what I've eaten?
How do you know? Better ability to cope with carbs, or did you have any tests done? I'm genuinely curious as I'm wondering if mine ever will ...Certainly my own insulin sensitivity continues to steadily improve.
How do you know? Better ability to cope with carbs, or did you have any tests done? I'm genuinely curious as I'm wondering if mine ever will ...
Well done, and without meds too! I'm in ketosis too. Last night I measured 1,3, but I'm still waiting for miracles (major weight loss) to happen. My BGs are mostly within 4,5 - 6,5, but would rather see them under 6 all the time. I'm still on metformin and 12ml of victoza, and wonder if that'll ever change, as I was diagnosed in 2005 + I'm an old bat too. Not complaining, though. I've done ok, everything taken into consideration.Just generally improving glucose stability. Irrespective of whether or not I’ve eaten, my glucose stays steady in a very narrow band all day long. Can’t specifically comment on carbohydrate as I don’t wish to consume any, but carbohydrate isn’t the only factor in determining blood glucose, and you don’t attain rock solid stability without a decent degree of insulin sensitivity
Well done, and without meds too! I'm in ketosis too. Last night I measured 1,3, but I'm still waiting for miracles (major weight loss) to happen. My BGs are mostly within 4,5 - 6,5, but would rather see them under 6 all the time. I'm still on metformin and 12ml of victoza, and wonder if that'll ever change, as I was diagnosed in 2005 + I'm an old bat too. Not complaining, though. I've done ok, everything taken into consideration.
Thanks for replying.
Have you ever considered that your problem all along might not have been insulin resistance but lack of insulin? Insulin resistance is often associated with being overweight but you were very light when diagnosed. If you now keep to very low carb diet then perhaps the insulin you can produce is sufficient to keep your BG under control. I think the jury is out on whether beta cells can recover when they are not being overused but that is another possibility.Thanks. Yes. I’m between 4.5-5.5mmol/L irrespective of food, and without any medications at all. This was from a position of chronic hyperglycaemia, diabetic retinopathy, macular edema, and peripheral neuropathy. Mostly I achieved this by intense self-education into the research of pioneers such as Jason Fung, Tim Noakes etc. so I’m naturally going to have the view that they know what they’re talking about
EDIT: current projected HbA1c is 29mmol/mol but I’m not sure I’ll be having another actual test anytime soon. Certainly not for another year or so. Takeaway lesson - listen to Jason Fung. The guy is a trailblazer in diabetes treatment
Thanks for a great reply Brunneria. I get what you mean about looking at a trend but isn't it good to also look out for what individual foods can cause a higher than normal post meal raise.
Does insulin resistance actually vary? Or is the insulin resistance a relatively constant thing with the glucose being the variant. Does the exercise affect how resistant your cells are to the insulin or are you just burning up more of the excess glucose produced or consumed?Absolutely!
But I just wouldn't take a single reaction to a single meal or food as 'gospel'.
I can get some really different readings for the same food, in the same portion sizes, depending on stuff like whether it was eaten early or late in the day (insulin resistance can vary during the day), the amount of stress and activity I have before and after... and so on.
As an example, I know perfectly well that a meal of pub burger and chips is going to affect my bg significantly. Probably spike me to 12+ on a normal day.
Not something I would eat regularly, although I have been known to do so.
I usually stick to extremely low (or no) carbs.
About 18 months ago we went on holiday and took a long, fairly strenuous walk along the Northumbrian coast, including exploring a castle, the tower, and clambering about. Significntly more exercise than usually fits into my sedentary(ish) life.
By the end of the walk, my legs had reached the wobbly stage, and my husband was carrying one of the dogs (haha!)
My bg normally runs in the 5s and 6s, but partway through that trek it dropped like a stone down into the low 4s and stayed there (presumably due to exercise induced reduced insulin resistance).
We ended up trundling cheerfully into a pub and I ate an ENORMOUS burger and chips. Delicious.
And my bg hardly shifted. I think it rose to the early 5s and stayed there for a while.
The next day, everything was back to normal, insulin resistance had bobbed back up like a cork, and I DIDN'T eat another burger and chips.
So yes, individual readings are always useful to know.
But they don't always add to your overall management. Better to do a few tests on a particular food or meal, in typical circumstances, and see what the general trend is.
I would love my 'typical circumstances' to include daily coastal walks and pub meals, but sadly I have a desk job, a fairly sedentary life, and my daily exercise is a dog walk at lunchtime on local streets or local parks. On the flat. With pauses for dog sniffs and chats to aquaintances.
You know, this subject is fascinating. I know diabetes is a terrible thing and obviously very dangerous to my health but I am really enjoying learning about it. You'll probably get sick of me asking questions soonDoes insulin resistance actually vary? Or is the insulin resistance a relatively constant thing with the glucose being the variant. Does the exercise affect how resistant your cells are to the insulin or are you just burning up more of the excess glucose produced or consumed?
Have you ever considered that your problem all along might not have been insulin resistance but lack of insulin? Insulin resistance is often associated with being overweight but you were very light when diagnosed. If you now keep to very low carb diet then perhaps the insulin you can produce is sufficient to keep your BG under control. I think the jury is out on whether beta cells can recover when they are not being overused but that is another possibility.
Does insulin resistance actually vary? Or is the insulin resistance a relatively constant thing with the glucose being the variant. Does the exercise affect how resistant your cells are to the insulin or are you just burning up more of the excess glucose produced or consumed?
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