NewdestinyX
Well-Known Member
- Messages
- 205
Not exactly, Pianoman. I agree we are way healthier than where we started - lowER carb to very lo carb diets predominantly work by simply lowering 'intake' of a whole macronutirent that turns directly into glucose in the blood - namely carbs.. So it's a no-brainer that low carbing can control BG levels.. The fact that BG numbers come into line doesn't prove anything except you've severly limited the main macronutrient (as an impetus) that elevates BG levels. But you have to keep doing that the rest of your life to keep numbers in line. That's no cure of any kind. It's a means to an end and a powerful one but only a controller of 'symptoms' methodology. The moment any of you increase your carbs -- numbers go right back up almost instantly as many low carbers have noted - the well known 'no-no foods' that will always spike.pianoman said:Except for the 'fact' that many (if not most) of those I converse with on these forums who are using a low-carb approach (including myself and dare I suggest you as well?) are doing so precisely because it has allowed them to manage improved (near normal) average monthly BGs and at least Pre-Diabetic A1c levels -- often with much reduced or no medication -- comparable to this study's results. By your own logic this suggests that they have indeed successfully reduced or eliminated excess fat from the affected organs.
What fat you lose when low carbing is from around your belly and though that reduces IR and lets the pancreas work more efficiently - which again helps the numbers game - it is not effective to 'cure' PP spikes after carby foods intake and not usually effective to alleviate most am fasting problems. In short - even fat loss from the adipose stores (the fat we carry) around the belly is often ineffective to cure poor First Phase insulin response - a chief attribute of T2D.
What this study claims and proved in a short term was to eliminate, SPECIFICALLY, liver and pancreas fat deposits - which is a NEW level of 'cure' in the sense that EVEN BEFORE your belly fat gets out of the way and reduces additional IR -- this 'de-fat-izing' of those two organs essentially does what the sulfonylureas attempt to do: MAKE the pancreas work more efficiently, nearly back to normal, improving First Phase Insulin response - so there's MORE insulin available to take care of ANY LEVEL of CARBS you eat - thus prevent spiking and improve fasting levels am and later in the day -(none of which am I claiming that this study absolutely accomplished -- rather I'm just making the point as to how comparing what low carbing does to what this study did is an 'apples and oranges' comparison).
What I described there is FUNDAMENTALLY a different level of 'diabetic control' than mere low carbing. It would, if effective, allow you to eat a more 'normal' level of carbs, that of a non-D person and not experience hyperglycemia as a symptom. Again that's a fundamentally DIFFERENT level of improvement than low carb could ever achieve. That's why it's so intriguing to me. Whether it WORKS long term is an entirely different matter. Low carbers are expert 'controllers/reducers of hyperglycemia' by severly limiting the main macronutrient that creates blood glucose level rises. This 600kcal 8 week diet attemtps to 'heal' the ALLEGED 'root' cause of the hyperglycemia (namely: fat around pancreas/liver) - and NOT just control the symptom -which is what low carb is. Low carbing to diabetes is akin to carrying an oxygen tank for reduced heart function. In both cases the pancreas and heart are no closer to working like they used to.