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<blockquote data-quote="Oldvatr" data-source="post: 1198014" data-attributes="member: 196898"><p>I think Richard Feinman. who is a leading antagonist for sugar in the American diet, would disagree with that.</p><p><a href="https://feinmantheother.com/2012/02/22/saturated-fat-on-your-plate-or-in-your-blood/" target="_blank">https://feinmantheother.com/2012/02/22/saturated-fat-on-your-plate-or-in-your-blood/</a></p><p></p><p>Recent reseach is showing that the SFA in our bloodstream is mostly self generated, and not directly related to dietary intake. In another thread we had a discussion (?) about overloaded FFA causing arterial damage, and I found that it seemed to be very difficult to actually produce overloaded FFA in vitro for experiments on rats. It is not something that seems to happen in real life. This was observed by Volek et al in their experiments.</p><p></p><p>If SFA in blood was so sticky, then we would not last very long. Our blood vessels are designed to carry this burden without problems through our lifetime. research seem to point out that damaged bloodvessels can trap detritus in the same way as a flooded rivers can build up silt at obstructions. The post mortem study has shown that the build up in plaque seems to be mainly damaged sdLDL and not FFA directly (i.e. it is the damaged lipid package, not its contents that is collecting).</p><p></p><p>Long chain SFA's are not entirely guilt free, but not for the reasons in your book:</p><p><a href="https://en.wikipedia.org/wiki/Very_long_chain_fatty_acid" target="_blank">https://en.wikipedia.org/wiki/Very_long_chain_fatty_acid</a></p></blockquote><p></p>
[QUOTE="Oldvatr, post: 1198014, member: 196898"] I think Richard Feinman. who is a leading antagonist for sugar in the American diet, would disagree with that. [URL]https://feinmantheother.com/2012/02/22/saturated-fat-on-your-plate-or-in-your-blood/[/URL] Recent reseach is showing that the SFA in our bloodstream is mostly self generated, and not directly related to dietary intake. In another thread we had a discussion (?) about overloaded FFA causing arterial damage, and I found that it seemed to be very difficult to actually produce overloaded FFA in vitro for experiments on rats. It is not something that seems to happen in real life. This was observed by Volek et al in their experiments. If SFA in blood was so sticky, then we would not last very long. Our blood vessels are designed to carry this burden without problems through our lifetime. research seem to point out that damaged bloodvessels can trap detritus in the same way as a flooded rivers can build up silt at obstructions. The post mortem study has shown that the build up in plaque seems to be mainly damaged sdLDL and not FFA directly (i.e. it is the damaged lipid package, not its contents that is collecting). Long chain SFA's are not entirely guilt free, but not for the reasons in your book: [URL]https://en.wikipedia.org/wiki/Very_long_chain_fatty_acid[/URL] [/QUOTE]
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