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Reactive Hypoglycemia
Additional diagnostic tests?
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<blockquote data-quote="Lamont D" data-source="post: 1970578" data-attributes="member: 85785"><p>Yeah I agree! It is something that has often had me looking for answers as for why my body is completely different to everyone else bar a few!</p><p></p><p></p><p>I'm different, because, my body used the excess insulin (hyperinsulinaemia) to make me gain weight. At just before diagnosis, I was over seventeen stone approaching eighteen and getting really ill. I have a photo of me, and I look as if I'm a balloon and been overblown.</p><p>Since diagnosis, the weight initially fell off and has been stable around 80 kilos for about four years now, which coincides with my good health and avoiding carbs.</p><p>Why did I increase weight when I ate very little?</p><p>The answer was insulin resistance. </p><p>With hyperinsulinaemia, the amount of insulin in my blood was very high, and because it never got used, it turned into visceral fat around my organs and midriff.</p><p>I had non alcoholic fatty liver, liver function, kidney function and high blood sugar levels. Because I was producing so much insulin, because my brain was getting the trigger to produce more due to the high glucose readings.</p><p>It was only when I started eating low carb, that the insulin resistance and blood sugar levels came down, hence being diagnosed as T2.</p><p>Once I reached normal hba1c levels and fasting levels, my endocrinologist, was intrigued at the difference in only a few weeks as I changed my diet.</p><p>He finally understood what was going on after reading my food diary and seeing me go hypo, at a couple of appointments.</p><p>More tests and more blood giving, lots of testing and experimentation!</p><p></p><p>My theory is there is something called a gut brain trigger, when we eat, all sorts of hormones and digestive chemicals from saliva to acids and of course glucose derived from the carbs and the insulin response, to what you have ate.</p><p>With RH, when too much glucose is derived, because of a weak initial insulin response, (the initial insulin response can be asymptomatic of T2 as well!) the spike triggers an overshoot of insulin, (a secondary response) this overshoot gets rid of the high blood glucose, but the insulin is too much now and causes the hypo.</p><p>So, that is why, no hyper, no hypo!</p><p>If you don't cause a spike, no secondary insulin response!</p><p>A lot of symptoms are to do with brain function, and the list is long and unhealthy.</p><p>The roller coaster ride of blood sugars is causing the symptoms, your brain has too much glucose, then not enough, then too much, then not enough and so on.</p><p>Then there is the other hormones like oestrogen in women, testosterone in men, thyroid, adrenaline, cortisol and more, that impact on how your system copes with its metabolic conditions.</p><p>Is it understandable that unless you have come across Hypoglycaemia in whatever form or type, that our medical profession have real difficulty in a diagnosis?</p><p>Or our personal GPs, who don't have the facilities or finance to get the necessary tests? That rely on their teachers to be aware of something that is common as a symptom but not as a condition?</p><p></p><p>I'm the only patient that has this condition in my surgery of over 2000 patients!</p><p>My endocrinologist has about thirty patients that have been diagnosed and referred to him. </p><p></p><p>Yes, it is an enigma, but, as I have found, treatment is more important than why!</p><p></p><p>Best wishes</p></blockquote><p></p>
[QUOTE="Lamont D, post: 1970578, member: 85785"] Yeah I agree! It is something that has often had me looking for answers as for why my body is completely different to everyone else bar a few! I'm different, because, my body used the excess insulin (hyperinsulinaemia) to make me gain weight. At just before diagnosis, I was over seventeen stone approaching eighteen and getting really ill. I have a photo of me, and I look as if I'm a balloon and been overblown. Since diagnosis, the weight initially fell off and has been stable around 80 kilos for about four years now, which coincides with my good health and avoiding carbs. Why did I increase weight when I ate very little? The answer was insulin resistance. With hyperinsulinaemia, the amount of insulin in my blood was very high, and because it never got used, it turned into visceral fat around my organs and midriff. I had non alcoholic fatty liver, liver function, kidney function and high blood sugar levels. Because I was producing so much insulin, because my brain was getting the trigger to produce more due to the high glucose readings. It was only when I started eating low carb, that the insulin resistance and blood sugar levels came down, hence being diagnosed as T2. Once I reached normal hba1c levels and fasting levels, my endocrinologist, was intrigued at the difference in only a few weeks as I changed my diet. He finally understood what was going on after reading my food diary and seeing me go hypo, at a couple of appointments. More tests and more blood giving, lots of testing and experimentation! My theory is there is something called a gut brain trigger, when we eat, all sorts of hormones and digestive chemicals from saliva to acids and of course glucose derived from the carbs and the insulin response, to what you have ate. With RH, when too much glucose is derived, because of a weak initial insulin response, (the initial insulin response can be asymptomatic of T2 as well!) the spike triggers an overshoot of insulin, (a secondary response) this overshoot gets rid of the high blood glucose, but the insulin is too much now and causes the hypo. So, that is why, no hyper, no hypo! If you don't cause a spike, no secondary insulin response! A lot of symptoms are to do with brain function, and the list is long and unhealthy. The roller coaster ride of blood sugars is causing the symptoms, your brain has too much glucose, then not enough, then too much, then not enough and so on. Then there is the other hormones like oestrogen in women, testosterone in men, thyroid, adrenaline, cortisol and more, that impact on how your system copes with its metabolic conditions. Is it understandable that unless you have come across Hypoglycaemia in whatever form or type, that our medical profession have real difficulty in a diagnosis? Or our personal GPs, who don't have the facilities or finance to get the necessary tests? That rely on their teachers to be aware of something that is common as a symptom but not as a condition? I'm the only patient that has this condition in my surgery of over 2000 patients! My endocrinologist has about thirty patients that have been diagnosed and referred to him. Yes, it is an enigma, but, as I have found, treatment is more important than why! Best wishes [/QUOTE]
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Additional diagnostic tests?
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