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Barry Groves -has he missed something?
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<blockquote data-quote="RussG" data-source="post: 213184" data-attributes="member: 30691"><p>Thanks Phoenix.</p><p></p><p>It is accurate to point out that the plural of anecdote is not data and thus it is dangerous to extrapolate. As for Pianoman's questions - these don't appear to be the basis of the trial. They were attempting to establish a biological pathway for T2 from obesity, at this stage irrespective of how obesity was achieved. AFAIK they were not looking at how one becomes fat, but suffice to say that a high fat diet did make the mice obese. No doubt there are other ways of achieving the same end point but they used a high fat diet here. Ergo it is accurate to say that a high fat diet in this experiment caused mice to become diabetic (as well as the other conclusions about the glucose sensing mechansim). That doesn't say that it is the <strong><u>only </u></strong>way to become diabetic.</p><p></p><p>FYI: here are the dietary breakdowns:</p><p></p><p></p><p><a href="http://www.nature.com/nm/journal/vaop/ncurrent/full/nm.2414.html#/methods" target="_blank">http://www.nature.com/nm/journal/vaop/ncurrent/full/nm.2414.html#/methods</a></p><p></p><p>Unless I've read it wrong, the point seems to be that becoming obese interrupts the mechansim for glucose / insulin homeostasis, i.e. the counter regulatory effect of glucose and insulin production / reception.</p><p></p><p>Given that not everyone who is obese becomes diabetic, and not everyone with (T2) diabetes is obese, this sounds like important research. I think your problem, guys, is with the newspaper report, not the science. It's not all about bashing low carb or promoting low fat, people.</p></blockquote><p></p>
[QUOTE="RussG, post: 213184, member: 30691"] Thanks Phoenix. It is accurate to point out that the plural of anecdote is not data and thus it is dangerous to extrapolate. As for Pianoman's questions - these don't appear to be the basis of the trial. They were attempting to establish a biological pathway for T2 from obesity, at this stage irrespective of how obesity was achieved. AFAIK they were not looking at how one becomes fat, but suffice to say that a high fat diet did make the mice obese. No doubt there are other ways of achieving the same end point but they used a high fat diet here. Ergo it is accurate to say that a high fat diet in this experiment caused mice to become diabetic (as well as the other conclusions about the glucose sensing mechansim). That doesn't say that it is the [b][u]only [/u][/b]way to become diabetic. FYI: here are the dietary breakdowns: [url]http://www.nature.com/nm/journal/vaop/ncurrent/full/nm.2414.html#/methods[/url] Unless I've read it wrong, the point seems to be that becoming obese interrupts the mechansim for glucose / insulin homeostasis, i.e. the counter regulatory effect of glucose and insulin production / reception. Given that not everyone who is obese becomes diabetic, and not everyone with (T2) diabetes is obese, this sounds like important research. I think your problem, guys, is with the newspaper report, not the science. It's not all about bashing low carb or promoting low fat, people. [/QUOTE]
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