You can have low C-peptide (lower end of normal range) and normal blood sugar means your insulin repsonse has been used up to normalise your blood sugars.Hi. I'm slightly surprised by this. How many people have a fasting glucose of 18mmol? I agree a mid-range C-Peptide doesn't tell you much. I would challenge that a low-C-Peptide and normal BS indicates normal insulin; it certainly doesn't in my case or anywhere near it. I wonder how many other universities such as Exeter would agree with the Oxford group? My second C-Peptide was a urine one analysed by Exeter and didn't involve measuring my fasting BS so how does that work?
Which is of course a classic T1 or LADA diagnosis.not producing my own insulin due to a worn out pancreas so feel ok about injecting.
So I could get 0.05 and still be making my own insulin?You can have low C-peptide (lower end of normal range) and normal blood sugar means your insulin has been used up to normalise your blood sugars.
I think the fact my daughters both have hyperinsulinism, dad was T2, mum T2, Sister T2, brother PreD, found out last night closest T1 is a Nan, strongly suggests a T2 with worn out pancreas to me. I’m ok with that because it makes sense to me.Which is of course a classic T1 or LADA diagnosis.
C-peptide is just a snapshot of your insulin. It's not a 3 month average like HbA1c. It needs to be interpreted with your fasting blood glucose. If your blood sugars are normal then you had an insulin response to bring them down.So I could get 0.05 and still be making my own insulin?
Did they ever test for antibodies? It’s not conclusive if negative but would be if positive.I think the fact my daughters both have hyperinsulinism, dad was T2, mum T2, Sister T2, brother PreD, found out last night closest T1 is a Nan, strongly suggests a T2 with worn out pancreas to me. I’m ok with that because it makes sense to me.
Isn’t it relevant what the blood sugars were (ie how far they had to come down) and how much insulin it took to do so? If the meal was very low carb for example and insulin resistance was minimal only a little insulin would be required and someone still producing a little insulin - for now - could cope and achieve a normal push bgl. Whereas a high carb meal with high starting bgl takes more insulin, and high IR would too, to achieve normality. Much easier to “fail” either due to underproduction or high IR.C-peptide is just a snapshot of your insulin. It's not a 3 month average like HbA1c. It needs to be interpreted with your fasting blood glucose. If your blood sugars are normal then you had an insulin response to bring them down.
Insulin resistance is when cells in your muscles, fat, and liver don't respond well to insulin and can't easily take up glucose from your blood. You're right. If you have a stimulated c-peptide it would give a better picture of this case.Isn’t it relevant what the blood sugars were (ie how far they had to come down) and how much insulin it took to do so? If the meal was very low carb for example and insulin resistance was minimal only a little insulin would be required and someone still producing a little insulin - for now - could cope and achieve a normal push bgl. Whereas a high carb meal with high starting bgl takes more insulin, and high IR would too, to achieve normality. Much easier to “fail” either due to underproduction or high IR.
or am I confusing everyone else as much as I am myself here. (My head is going in circles tonight) Someone hopefully will understand my gibberish and translate.
Do you know your results? Are you happy to have that diagnosis?So I did get referred for the c-peptide and GAD shortly after I posted this and got the results back this week. I have been diagnosed with late-onset type 1 (26) Xx
Do you know your results? Are you happy to have that diagnosis?
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