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Caffeine causes insulin release?
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<blockquote data-quote="Oldvatr" data-source="post: 2292455" data-attributes="member: 196898"><p>To my mind their observations on fatty acids being increased by coffee or caffein is of more importance than thefasting glucose levels.</p><p></p><p>Reading between the lines the second study seems to show that while glucose was relatively unchanged, the insulin levels rose, as did the free fatty acids. Is there a connection there? They moot a theory about Insulin resistance, but to my mind it actually points to only one of the two IR modes being affected, namely muscle IR did not change (glucose was stored ok) but insulin did not drop as much implying it was not being used or stored in the adipose tissues, i.e. hepatic or adipose IR.</p><p></p><p>This would be in line with the Newcastle Diet findings of Prof Taylor which to my mind only addressed the adipose IR aspect. </p><p>Importantly it would seem that different mechanism are at play and thus full remission for T2D will require a two pronged attack.</p></blockquote><p></p>
[QUOTE="Oldvatr, post: 2292455, member: 196898"] To my mind their observations on fatty acids being increased by coffee or caffein is of more importance than thefasting glucose levels. Reading between the lines the second study seems to show that while glucose was relatively unchanged, the insulin levels rose, as did the free fatty acids. Is there a connection there? They moot a theory about Insulin resistance, but to my mind it actually points to only one of the two IR modes being affected, namely muscle IR did not change (glucose was stored ok) but insulin did not drop as much implying it was not being used or stored in the adipose tissues, i.e. hepatic or adipose IR. This would be in line with the Newcastle Diet findings of Prof Taylor which to my mind only addressed the adipose IR aspect. Importantly it would seem that different mechanism are at play and thus full remission for T2D will require a two pronged attack. [/QUOTE]
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