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<blockquote data-quote="Member496333" data-source="post: 2163316"><p>I love you but if I read this one more time my head might explode <img src="data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7" class="smilie smilie--sprite smilie--sprite39" alt=":hilarious:" title="Hilarious :hilarious:" loading="lazy" data-shortname=":hilarious:" /></p><p></p><p>It is true that GNG is demand driven, but if you're not getting the balance of fuel from fat then there IS a demand for glucose and the liver will make it from amino acids if it needs to. I suspect the amount of body fat that one has available to burn (or is able to burn*) is a major deciding factor in whether or not the liver turns to manufacturing glucose. *Pre-existing hyperinsulinemia may also hinder ketone production, in turn forcing the liver to turn to glucose in situations where fuel is scarce.</p><p></p><p>Things will eventually settle down as insulin sensitivity returns, but those with very little body fat may still find that they need to be mindful of getting enough fuel from fat, otherwise their body will need to turn to glucose production, and excess amino acids give it the raw materials with which to do so.</p><p></p><p>I don't mean to sound argumentative. I respect your views greatly, and you are right on the whole, but the protein story is definitely a little more nuanced than many might realise. As a TOFI who once had major problems with it, you can be sure that I've done exhausting research and self-experimentation. In short, it is entirely possible that some people - particular diabetics with little fat to spare - may experience raised glucose when protein is abundant and dietary fat is too low. </p><p></p><p>Now where's that bacon? <img src="data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7" class="smilie smilie--sprite smilie--sprite8" alt=":D" title="Big Grin :D" loading="lazy" data-shortname=":D" /></p></blockquote><p></p>
[QUOTE="Member496333, post: 2163316"] I love you but if I read this one more time my head might explode :hilarious: It is true that GNG is demand driven, but if you're not getting the balance of fuel from fat then there IS a demand for glucose and the liver will make it from amino acids if it needs to. I suspect the amount of body fat that one has available to burn (or is able to burn*) is a major deciding factor in whether or not the liver turns to manufacturing glucose. *Pre-existing hyperinsulinemia may also hinder ketone production, in turn forcing the liver to turn to glucose in situations where fuel is scarce. Things will eventually settle down as insulin sensitivity returns, but those with very little body fat may still find that they need to be mindful of getting enough fuel from fat, otherwise their body will need to turn to glucose production, and excess amino acids give it the raw materials with which to do so. I don't mean to sound argumentative. I respect your views greatly, and you are right on the whole, but the protein story is definitely a little more nuanced than many might realise. As a TOFI who once had major problems with it, you can be sure that I've done exhausting research and self-experimentation. In short, it is entirely possible that some people - particular diabetics with little fat to spare - may experience raised glucose when protein is abundant and dietary fat is too low. Now where's that bacon? :D [/QUOTE]
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