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Message: <blockquote data-quote="jddukes" data-source="post: 2650123" data-attributes="member: 49946">Thanks for starting this topic. I was always under the naive and erroneous assumption that "dawn phenomenon" (DP) was a gluconeogenesis in the liver phenomenon, purely. I didn't feel I really suffered from it.Then more recently, I have switched to a carnivore diet (on top of intermittent fasting where I don't eat before 13:00 every day), so have 0 carbs, essentially (certainly <10g in a day), and no food in the morning. I previously have done keto diets (not carnivore), and not observed DP then, but realise I was on Lantus then. I am now on Levemir.The DP I see, where I wake up and my levels might be at 8-9mmol/l, and as soon as I get up they shoot up to 11-12mmol/l. This is happening without carbs in the evening (although I wonder if too much protein is contributing here), and instantly on getting out of bed it starts to go up. Work has been quite intense personally and I've put this down to increased cortisol on waking - cortisol is known to increase BS levels. However, I would hope (expect?) that longer term carnivore/very low carb diets would deplete glycogen store availability to reduce the effect of cortisol (I struggle with the idea of gluconeogenesis occurring so rapidly on waking, but perhaps there is the sleeping pattern regularity that my body is operating under and so as I wake up the same time each day pretty much, this is happening earlier and snowballs?).So I'm guessing the best solution for this in the near term sounds from the advice here to just instantly take some novorapid on waking (this can be a challenge, as I don't eat, and then often cycle to work which means novorapid + fasted state + exercise = large effect of reducing BS levels risking hypos following the exercise), but perhaps pursuing the pump may be a good way to manage this?</blockquote>

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