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Type 1 Diabetes
Fixed doeses and Lantus dose
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<blockquote data-quote="paulus1" data-source="post: 1627590" data-attributes="member: 191134"><p>well that explains the jump spuds are very heavy in carbs. perhaps a little more meat. what was his night time reading. if over 6 you not need to feed him. unless low say in the 4s leave off the bread. remember we are trying to not spike the bg too much. his diet can be as normal has he wants but some food will raise it. he will learn what over time.</p><p></p><p>ok type</p><p>type 1 is normally seen in children and teens some older people can develop it. usually high bg and ketones</p><p>type 1.5 or lada usual develops in 30-50 year olds is slower onset than 1 and may resemble type 2</p><p>type 2 is mainly found in 40 plus caused by insulin intolerance it is found now in some obese teens and children. type 2 can have high bg but tend not to have ketones. </p><p>type3 not your son its Alzheimer’s disease</p><p>type 3c injury/surgery or pancreatic illnesses cause the death or removal of the insulin producing cells. </p><p>Maturity Onset Diabetes of the Young affects 1-2%of diabetics it runs in families </p><ul> <li data-xf-list-type="ul"><span style="color: #ff0000">Mild to moderate hyperglycemia (typically 130–250 mg/dl, or 7–14 mmol/l)</span> discovered before 30 years of age. However, anyone under 50 can develop MODY.<a href="https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-10" target="_blank">[10]</a></li> <li data-xf-list-type="ul"><span style="color: #ff0000">A first-degree relative with a similar degree of diabetes.</span></li> <li data-xf-list-type="ul">Absence of positive antibodies or other <a href="https://en.wikipedia.org/wiki/Autoimmunity" target="_blank">autoimmunity</a> (e.g., <a href="https://en.wikipedia.org/wiki/Thyroiditis" target="_blank">thyroiditis</a>) in patient and family. However, Urbanova et al. found that about one quarter of Central European MODY patients are positive for islet cell autoantibodies (GADA and IA2A). Their expression is transient but highly prevalent. The autoantibodies were found in patients with delayed diabetes onset, and in times of insufficient diabetes control. The islet cell autoantibodies are absent in MODY in at least some populations (Japanese, Britons).<a href="https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-11" target="_blank">[11]</a></li> <li data-xf-list-type="ul">Persistence of a low insulin requirement (e.g., less than 0.5 u/kg/day) past the usual "<a href="https://en.wikipedia.org/wiki/Honeymoon_period_(diabetes)" target="_blank">honeymoon</a>" period.</li> <li data-xf-list-type="ul">Absence of obesity (although overweight or obese people can get MODY) or other problems associated with type 2 diabetes or <a href="https://en.wikipedia.org/wiki/Metabolic_syndrome" target="_blank">metabolic syndrome</a> (e.g., <a href="https://en.wikipedia.org/wiki/Hypertension" target="_blank">hypertension</a>, <a href="https://en.wikipedia.org/wiki/Hyperlipidemia" target="_blank">hyperlipidemia</a>, <a href="https://en.wikipedia.org/wiki/Polycystic_ovary_syndrome" target="_blank">polycystic ovary syndrome</a>).<a href="https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-12" target="_blank">[12]</a></li> <li data-xf-list-type="ul"><a href="https://en.wikipedia.org/wiki/Insulin_resistance" target="_blank">Insulin resistance</a> very rarely happens.<a href="https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-13" target="_blank">[13]</a></li> <li data-xf-list-type="ul">Cystic <a href="https://en.wikipedia.org/wiki/Kidney" target="_blank">kidney</a> disease in patient or close relatives.</li> <li data-xf-list-type="ul">Non-transient <a href="https://en.wikipedia.org/wiki/Neonatal_diabetes" target="_blank">neonatal diabetes</a>, or apparent <a href="https://en.wikipedia.org/wiki/Type_1_diabetes_mellitus" target="_blank">type 1 diabetes</a> with onset before six months of age.</li> <li data-xf-list-type="ul">Liver adenoma or hepatocellular carcinoma in MODY type 3<a href="https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-14" target="_blank">[14]</a></li> <li data-xf-list-type="ul">Renal cysts, rudimentary or bicornuate uterus, vaginal aplasia, absence of the vas deferens, epidymal cysts in MODY type 5<a href="https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-15" target="_blank">[15]</a></li> </ul><p>The diagnosis of MODY is confirmed by specific <a href="https://en.wikipedia.org/wiki/Gene" target="_blank">gene</a> testing available through commercial laboratories.</p><p></p><p>these are the main types to confirm if its type 1 or lada a gad test is done it takes a few weeks to come back. mody requires a dna test and takes awhile. of the types 2 effects 90% of diabetics. type1about 10% mody and lada below 5% each.</p><p></p><p>we really can discount type 2 he does not fit the pattern. he fits type 1 does not have lada to young. mody is rare and usually is lower your sons 30 reading does not really go along with it. if there are no others in your family with mody its unlikely.</p><p></p><p>So your diabetic consultant will deal with the main issue and thats what they have done. its most likely to be type 1 so thats the treatment. later they will do the tests for antibodies to confirm it. if not they will test for mody. but for now he needs this treatment. the insulin will do him no harm even if he was mody and does not need insulin in the future. </p><p></p><p>i hope this helps im not trying to lecture you. sorry if it comes out that way.</p></blockquote><p></p>
[QUOTE="paulus1, post: 1627590, member: 191134"] well that explains the jump spuds are very heavy in carbs. perhaps a little more meat. what was his night time reading. if over 6 you not need to feed him. unless low say in the 4s leave off the bread. remember we are trying to not spike the bg too much. his diet can be as normal has he wants but some food will raise it. he will learn what over time. ok type type 1 is normally seen in children and teens some older people can develop it. usually high bg and ketones type 1.5 or lada usual develops in 30-50 year olds is slower onset than 1 and may resemble type 2 type 2 is mainly found in 40 plus caused by insulin intolerance it is found now in some obese teens and children. type 2 can have high bg but tend not to have ketones. type3 not your son its Alzheimer’s disease type 3c injury/surgery or pancreatic illnesses cause the death or removal of the insulin producing cells. Maturity Onset Diabetes of the Young affects 1-2%of diabetics it runs in families [LIST] [*][COLOR=#ff0000]Mild to moderate hyperglycemia (typically 130–250 mg/dl, or 7–14 mmol/l)[/COLOR] discovered before 30 years of age. However, anyone under 50 can develop MODY.[URL='https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-10'][10][/URL] [*][COLOR=#ff0000]A first-degree relative with a similar degree of diabetes.[/COLOR] [*]Absence of positive antibodies or other [URL='https://en.wikipedia.org/wiki/Autoimmunity']autoimmunity[/URL] (e.g., [URL='https://en.wikipedia.org/wiki/Thyroiditis']thyroiditis[/URL]) in patient and family. However, Urbanova et al. found that about one quarter of Central European MODY patients are positive for islet cell autoantibodies (GADA and IA2A). Their expression is transient but highly prevalent. The autoantibodies were found in patients with delayed diabetes onset, and in times of insufficient diabetes control. The islet cell autoantibodies are absent in MODY in at least some populations (Japanese, Britons).[URL='https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-11'][11][/URL] [*]Persistence of a low insulin requirement (e.g., less than 0.5 u/kg/day) past the usual "[URL='https://en.wikipedia.org/wiki/Honeymoon_period_(diabetes)']honeymoon[/URL]" period. [*]Absence of obesity (although overweight or obese people can get MODY) or other problems associated with type 2 diabetes or [URL='https://en.wikipedia.org/wiki/Metabolic_syndrome']metabolic syndrome[/URL] (e.g., [URL='https://en.wikipedia.org/wiki/Hypertension']hypertension[/URL], [URL='https://en.wikipedia.org/wiki/Hyperlipidemia']hyperlipidemia[/URL], [URL='https://en.wikipedia.org/wiki/Polycystic_ovary_syndrome']polycystic ovary syndrome[/URL]).[URL='https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-12'][12][/URL] [*][URL='https://en.wikipedia.org/wiki/Insulin_resistance']Insulin resistance[/URL] very rarely happens.[URL='https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-13'][13][/URL] [*]Cystic [URL='https://en.wikipedia.org/wiki/Kidney']kidney[/URL] disease in patient or close relatives. [*]Non-transient [URL='https://en.wikipedia.org/wiki/Neonatal_diabetes']neonatal diabetes[/URL], or apparent [URL='https://en.wikipedia.org/wiki/Type_1_diabetes_mellitus']type 1 diabetes[/URL] with onset before six months of age. [*]Liver adenoma or hepatocellular carcinoma in MODY type 3[URL='https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-14'][14][/URL] [*]Renal cysts, rudimentary or bicornuate uterus, vaginal aplasia, absence of the vas deferens, epidymal cysts in MODY type 5[URL='https://en.wikipedia.org/wiki/Maturity_onset_diabetes_of_the_young#cite_note-15'][15][/URL] [/LIST] The diagnosis of MODY is confirmed by specific [URL='https://en.wikipedia.org/wiki/Gene']gene[/URL] testing available through commercial laboratories. these are the main types to confirm if its type 1 or lada a gad test is done it takes a few weeks to come back. mody requires a dna test and takes awhile. of the types 2 effects 90% of diabetics. type1about 10% mody and lada below 5% each. we really can discount type 2 he does not fit the pattern. he fits type 1 does not have lada to young. mody is rare and usually is lower your sons 30 reading does not really go along with it. if there are no others in your family with mody its unlikely. So your diabetic consultant will deal with the main issue and thats what they have done. its most likely to be type 1 so thats the treatment. later they will do the tests for antibodies to confirm it. if not they will test for mody. but for now he needs this treatment. the insulin will do him no harm even if he was mody and does not need insulin in the future. i hope this helps im not trying to lecture you. sorry if it comes out that way. [/QUOTE]
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