Glucagon Admittance and the Central Equation of Diabetes

Dear all,

A few years ago I posted on "The Cure" for type two diabetes. While we did get a few headline cures most people did not get better. Many people enjoyed dramatic weight loss though.

By way of appology to those who did not cure I resolved to find out why and I have done so. Not only that I have produced a very promising model of type two diabetes which I believe solves the so called "central problem of diabetes". The following link leads you to a couple of files: The first is a powerpoint slideshow which you should SAVE rather than OPEN before running. For a reason as yet unknown powerpoint seems unable to produce a stand alone slideshow - advice here would be welcome. The second "Word" post is closely based on a letter to a diabetic nurse which explains how the modified electron transport chain leads to four different sub-types of diabetes and which of the sub-types are cureable.

http://www.glucagonadmittance.com

For future reference there is a new treatment out called "Pancreate". This should work for genetic types who are not cureable via "The Cure". I'm not sure when it will be available.

The slideshow is under development and more graphs etc. are planned.

Hope this helps.


JM

Dear Hobs,

Thanks for the information. The site is in the charge of a third party. I will forward your advice.

All please note that the model is a proposal. It is well known that diabetes is caused by trans-fatty acids and the modified electron transport chain is in the literature. What I have tried to do is propose a method by which the set point is established. This done the model predicts that as poisoning continues the set point of the control mechanism rises so that the pancrease tries to regulate blood sugar to progressively higher and higher levels. In the terminal stages even taking full medication and fasting for days will still leave you with very high blood sugar. The glucose in your blood is not as a result of what you have eaten but as a result of excess glucagon production. You are making your own blood sugar! Kings College have proposed that this is as a result of a blocking of the glucagon inhibitor my suggestion is that it is as a result of different bundle sizes meaning that the glucagon is switched off later than the insulin is switched on and this difference increases as poisoning progresses.

My model needs further development and referencing and doubltless modification.

JM