Yes, sadly my experience of diabetes GPs is the same. They can be good general GPs but when it comes to diabetes they tend to have known less than me as the training appears to be very poor (like never heard of Late onset T1 i.e. LADA). That sounds arrogant but sadly true in my experience. My DN is much better she knows her limits but does a good job with what she does know and listens and questions. So be prepared to use your own knowledge bank when needed.Every living human experiences the dawn phenomenon. It’s a result of the endocrine system releasing hormones that signal the liver to secrete glucose in order to help us wake from sleep and start the day. The measurable increases in blood glucose concentration in diabetics comes about because their insulin is largely ineffective at facilitating the uptake of that glucose into the cells, and because their insulin resistant liver doesn’t stop releasing it.
I should be surprised that a diabetes care professional doesn’t know this, but I’m not. In fact in my experience most diabetes professionals are simply reading from a pre-approved list of official advice and will dismiss anything you say as codswallop. After all, their knowledge of the disease is vastly superior
EDIT: my description is relating to type 2, but obviously the DP mechanism itself is common to all humans. I’m unsure how it interacts with other forms of diabetes.
A few years ago my surgery stopped all fasting glucose tests completely, even on non-diabetics at routine MOT tests. They only do an HbA1c.
I was told the reason for this was because of the unreliability due to raised levels caused by glucose dumps from the liver in some people, exacerbated by stress at getting to the surgery and anxiety about the forthcoming tests. So at least my surgery understands this!
Not sure I agree entirely with that logic. A metabolically healthy person shouldnt really exhibit big spikes. If they do then they’re insulin resistant. Whilst I understand the sentiment, one can make an argument that it allows more people with insulin resistance to slip through the net and “score” a HbA1c sufficiently averaged to avoid a diagnosis.
Happy to disagree though of course
Hi @Belzedar. Somogyi effect and DP are separate things. You would have to prove that there were low BSLs occurring with a related rebound BSL rise. People get DP for different reasons than Somogyi. Please get this straight otherwise you will be confused.Thanks @kitedoc These posts have been very thought-provoking and quite helpful. In fact, I hit on a new line of enquiry that really resonates with me. The Dawn Phenomenon is universal. It’s the presence of cortisol and Human Growth Hormone readying the body for action. The liver plays its part by using neoglucogenisis to dump glucose into the blood for transport round the body. Everything is doing its job.
Now, the pancreas is doing its job by releasing pulses of basal insulin every couple of minutes to keep the liver from overshooting. BUT if the liver is insulin resistant, it’s incapable of responding to that and carries on regardless - dumping glucose into the blood. This, I've read, happens whether the liver responds to low sugar levels after several hours of fasting during sleep (the Somagyi effect) end even in the “fed” state (because of the hepatic insulin resistance). I had recently discovered the effectiveness of taking a shot of Novorapid at about 3am to anticipate the glucose spike from 4am and it was this that my Doctor and I parted ways. She forbade me to ever taking insulin without food - without pausing for a second to consider that I wasn’t susceptabile to hypos, having had less than twenty recorded hypo readings in the past seven years (yup, that’s about two or three low readings a year) and for my part MORE more carbs just to satisy her requirement wasn’t likely to bring my glucose levels
A diabetic can suffer big BSL spikes for a number of reasons e.g. eating many carbs at a meal. In that case the BSL spike from this is NOT due to insulin resistance.Not sure I agree entirely with that logic. A metabolically healthy person shouldnt really exhibit big spikes. If they do then they’re insulin resistant. Whilst I understand the sentiment, one can make an argument that it allows more people with insulin resistance to slip through the net and “score” a HbA1c sufficiently averaged to avoid a diagnosis.
Happy to disagree though of course
What is so?I said a metabolically healthy person, in the context of blood checks to determine whether or not that is so
What is so?
Please define "metabolic issues".The discussion at the time of my post was regarding blood tests to determine whether or not a person has any metabolic issues. Not whether or not diabetics exhibit dawn phenomenon and why.
Ok, but I am in the Southern hemisphere so bedtime is a tad different. Insulin resistance is not actually that easy to measure that is whyI was trying to understand you.In this context - insulin resistance, insulin deficiency, diabetes. Not sure where you’re going with this but it seems you may need to go back to bed and get out the other side. I’m not looking for an argument.
Have a great day
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