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<blockquote data-quote="kokhongw" data-source="post: 1359172" data-attributes="member: 277199"><p>[USER=370633]@Confucius[/USER] I posted this on another thread, but this it may also be relevant here.</p><p></p><p>The current mainstream view of T2D is very glucose centric. We are looking only at the glucose side of the problem...simply because it can be tested easily and readily.</p><p></p><p>What most of us are not aware it that impaired fasting glucose for T2D is preceded by years of elevated insulin levels commonly due to high carbs/fructose diet. That is the key reason for the beta cells dysfunction and exhaustion by the time of T2D diagnosis.</p><p></p><p>So while our numbers may not be clinically "diabetic", we already have impaired insulin/glucose response. The guide for clinical diagnosis is centered on the necessity for prescription medication to reduce obvious/immediate complications due to high glucose level. The mainstream approach does little to nothing for restoring the beta cells dysfunction. Understanding this will save us years of grief.</p><p></p><p>Dr Jason Fung's recent discussion on this will give some addtional insights on why a carbs lite fats friendly approach is often effective for halting the progression of T2D, allowing some recovery. Because dietary fats requires the least insulin response.</p><p></p><p><a href="https://intensivedietarymanagement.com/understanding-joseph-kraft-diabetes-in-situ-t2d-24/" target="_blank">https://intensivedietarymanagement.com/understanding-joseph-kraft-diabetes-in-situ-t2d-24/</a></p><p><img src="https://intensivedietarymanagement.com/wp-content/uploads/2016/12/Kraft-Curves-Cummins.png" alt="" class="fr-fic fr-dii fr-draggable " style="" /></p></blockquote><p></p>
[QUOTE="kokhongw, post: 1359172, member: 277199"] [USER=370633]@Confucius[/USER] I posted this on another thread, but this it may also be relevant here. The current mainstream view of T2D is very glucose centric. We are looking only at the glucose side of the problem...simply because it can be tested easily and readily. What most of us are not aware it that impaired fasting glucose for T2D is preceded by years of elevated insulin levels commonly due to high carbs/fructose diet. That is the key reason for the beta cells dysfunction and exhaustion by the time of T2D diagnosis. So while our numbers may not be clinically "diabetic", we already have impaired insulin/glucose response. The guide for clinical diagnosis is centered on the necessity for prescription medication to reduce obvious/immediate complications due to high glucose level. The mainstream approach does little to nothing for restoring the beta cells dysfunction. Understanding this will save us years of grief. Dr Jason Fung's recent discussion on this will give some addtional insights on why a carbs lite fats friendly approach is often effective for halting the progression of T2D, allowing some recovery. Because dietary fats requires the least insulin response. [URL]https://intensivedietarymanagement.com/understanding-joseph-kraft-diabetes-in-situ-t2d-24/[/URL] [IMG]https://intensivedietarymanagement.com/wp-content/uploads/2016/12/Kraft-Curves-Cummins.png[/IMG] [/QUOTE]
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