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Ivor Cummins in keto debate
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<blockquote data-quote="Sean_Raymond" data-source="post: 2266601" data-attributes="member: 403497"><p>The reason a high insulin state would not be expected to produce a drive to eat is because, as suggested by the body of evidence, Insulin exerts anorexigenic effects rather than orexigenic ones. Indeed, an interesting recent study showed that when reducing blood sugars beyond the starting BM (after administration of glucose) from insulin release no impact on hunger was seen. The study indicates that reducing blood sugars in a way that mimicked the so called post meal blood sugar spike and crash did not stimulate appetite. 1 study but an interesting one seeming to confirm that in the absence of a frank hypo spikes/drops in blood sugar do not stimulate a desire to eat. <a href="https://www.sciencedirect.com/science/article/abs/pii/S0195666316302604" target="_blank">https://www.sciencedirect.com/science/article/abs/pii/S0195666316302604</a></p><p></p><p>Conclusive evidence does not exist that insulin increases food intake rather it indicates that it is the opposite way around with a manipulation of insulin levels not producing a drive to eat. Adding in a state of insulin resistance would not change this – whether a study directly looking at the effect of a hyperinsuliaemic insulin resistant state on appetite exists I’m not sure of but if it does not this doesn’t disprove anything I have said.</p><p></p><p>Hmmnnnnn…you have a disease which has caused inflammation in your joints – this inflammation hasn’t gone away because you do not move to minimise pain. Not moving may mask the condition but this merely helps you cope with it rather than fix it. I don’t think this example accurately represents what we are discussing anyway.</p><p></p><p>We agree that a hypo stimulates hunger – we seem to disagree about where the fault of insulin lies in this. We also agree it is pointless to discuss this further J. I find it always interesting to have such discussions so cheers.</p></blockquote><p></p>
[QUOTE="Sean_Raymond, post: 2266601, member: 403497"] The reason a high insulin state would not be expected to produce a drive to eat is because, as suggested by the body of evidence, Insulin exerts anorexigenic effects rather than orexigenic ones. Indeed, an interesting recent study showed that when reducing blood sugars beyond the starting BM (after administration of glucose) from insulin release no impact on hunger was seen. The study indicates that reducing blood sugars in a way that mimicked the so called post meal blood sugar spike and crash did not stimulate appetite. 1 study but an interesting one seeming to confirm that in the absence of a frank hypo spikes/drops in blood sugar do not stimulate a desire to eat. [URL]https://www.sciencedirect.com/science/article/abs/pii/S0195666316302604[/URL] Conclusive evidence does not exist that insulin increases food intake rather it indicates that it is the opposite way around with a manipulation of insulin levels not producing a drive to eat. Adding in a state of insulin resistance would not change this – whether a study directly looking at the effect of a hyperinsuliaemic insulin resistant state on appetite exists I’m not sure of but if it does not this doesn’t disprove anything I have said. Hmmnnnnn…you have a disease which has caused inflammation in your joints – this inflammation hasn’t gone away because you do not move to minimise pain. Not moving may mask the condition but this merely helps you cope with it rather than fix it. I don’t think this example accurately represents what we are discussing anyway. We agree that a hypo stimulates hunger – we seem to disagree about where the fault of insulin lies in this. We also agree it is pointless to discuss this further J. I find it always interesting to have such discussions so cheers. [/QUOTE]
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