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<blockquote data-quote="JohnEGreen" data-source="post: 2286934" data-attributes="member: 223921"><p>[USER=94045]@Bluetit1802[/USER] I almost certainly know less than your self about this I was just trying to find out about how my pottasium levels would effect my diabetes.</p><p></p><p>I just found this on my travels not much detail.</p><p></p><p>"If your potassium levels are too low, your body may make less insulin. That could lead to high blood sugar. Studies show that people with low potassium levels release less insulin, have higher blood sugar levels, and are more likely to get type 2 diabetes than those with normal potassium levels.</p><p>There isn't enough evidence to prove that low potassium directly causes diabetes. Still, it might be a good idea to have your doctor check your potassium level if you're already at risk for diabetes."</p><p></p><p><a href="https://www.webmd.com/diabetes/potassium-diabetes#:~:text=If%20your%20potassium%20levels%20are,those%20with%20normal%20potassium%20levels" target="_blank">https://www.webmd.com/diabetes/potassium-diabetes#:~:text=If your potassium levels are,those with normal potassium levels</a>.</p><p></p><p>And then this.</p><p></p><p>"All cells in the body maintain an uneven distribution of electric charges—mostly carried by ions such as potassium, sodium, and chloride—across their outer membranes, which are therefore polarized. Changes in membrane polarization act as signals for various cell functions. In β-cells, a reduction of the transmembrane charge difference, called depolarization, triggers insulin release. The molecule that links membrane polarization to insulin release is the ATP-dependent potassium channel. The channel sits at the β-cell's outer membrane and keeps the membrane polarized by maintaining a sharp gradient of potassium distribution across the membrane. As long as the membrane is polarized, β-cells keep insulin trapped inside secretory vesicles. But as β-cells take up glucose, they transform the sugar into ATP, a small energy-carrying molecule that closes the potassium channel. The resulting membrane depolarization causes a massive influx of calcium inside the cells, which in turn allows the vesicles to release insulin to the outside."</p><p></p><p><a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1334240/" target="_blank">https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1334240/</a></p></blockquote><p></p>
[QUOTE="JohnEGreen, post: 2286934, member: 223921"] [USER=94045]@Bluetit1802[/USER] I almost certainly know less than your self about this I was just trying to find out about how my pottasium levels would effect my diabetes. I just found this on my travels not much detail. "If your potassium levels are too low, your body may make less insulin. That could lead to high blood sugar. Studies show that people with low potassium levels release less insulin, have higher blood sugar levels, and are more likely to get type 2 diabetes than those with normal potassium levels. There isn't enough evidence to prove that low potassium directly causes diabetes. Still, it might be a good idea to have your doctor check your potassium level if you're already at risk for diabetes." [URL]https://www.webmd.com/diabetes/potassium-diabetes#:~:text=If%20your%20potassium%20levels%20are,those%20with%20normal%20potassium%20levels[/URL]. And then this. "All cells in the body maintain an uneven distribution of electric charges—mostly carried by ions such as potassium, sodium, and chloride—across their outer membranes, which are therefore polarized. Changes in membrane polarization act as signals for various cell functions. In β-cells, a reduction of the transmembrane charge difference, called depolarization, triggers insulin release. The molecule that links membrane polarization to insulin release is the ATP-dependent potassium channel. The channel sits at the β-cell's outer membrane and keeps the membrane polarized by maintaining a sharp gradient of potassium distribution across the membrane. As long as the membrane is polarized, β-cells keep insulin trapped inside secretory vesicles. But as β-cells take up glucose, they transform the sugar into ATP, a small energy-carrying molecule that closes the potassium channel. The resulting membrane depolarization causes a massive influx of calcium inside the cells, which in turn allows the vesicles to release insulin to the outside." [URL]https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1334240/[/URL] [/QUOTE]
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