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<blockquote data-quote="Oldvatr" data-source="post: 2306980" data-attributes="member: 196898"><p>If I had evidence of, or suspected tht, by taking a chemical from Food(A) and mixing it with a chemical from Food(B) to make a new chemical (C) that had the power to remove adipose fat without disturbing the rest of the lymphatic system, then I would keep stumm about it until I had isolated the process, managed to synthesise it in a testube, make sure I could scale up to making large vats of it in a factory. Then I would make sure I patented it, This has got to be the Golden Goose of this century, and I would not faff about selling diet plans and leaving it open to others to make their fortunes from. Is this why explanations in the website are so poor and insubstantial?</p><p></p><p>I am having real difficulty seeing how this diet would lead to the claims being made for it especially for T2 diabetics. It is unrestricted calories, and one can eat as much as one wants to. It allows foods such as oatmeal, brown rice, quinoa, and bread It is certainly not Keto, nor VLCD, but is very, very low fat. The high level of carbs recommended in unlimited amounts and uncontrolled portion sizes is an anathema to most T2D reading this and it defies both science (carbs directly lead to increasing glucose levels) and personal experiences, and anecdotal evidence throughout this Forum. Even normal non diabetics have a rise in glucose levels when eating carbs. The body is designed to respond to the amylase rushes to give two insulin responses (but T2D have an impaired reaction) and this is how it has been since man first walked the earth.</p><p></p><p>So my first stake in the ground is that a high carb diet will result in a rise in bgl. It is nothing to do with omnivore vs plant based, but is due to diets with high carb intake. </p><p></p><p>Moving on and assuming this stake remains standing, then a T2D eating some foods as recommended will see their bgl start to tise after the meal. Since one hallmark of T2D is insulin resistancem then this will mean that the body has difficulty in either using up or storing the glucose in the blood, and it will tend to remain atelevted levels for some time. This level of bgl is enough to prevent not just storage but also of using stored glucose until bgl levels drop to normal. Importantly it stops the body creating glucose from lipids or emptying the existing glucose stores in the liver. So it keeps what you already got. Adipose fat remains untouched.</p><p></p><p>Gradually bgl levels drop as we expend energy. Since the diet states that exercise is not a requirement of the diet plan then this may take a while. Now considering the adipose fat (tiny lipid particles as the diet states) then this only gets used when the body is not in storage mode, so having high levels of insulin and high glucose levels prevents the body from using lipids as a source of energy. So the average T2D will spend most of the day trying to force excess glucose into storage, which is the hallmark of our condition.</p><p></p><p>But this diet has one thing in its favour. It is ultra low fat (nuts anybody?). This means that the overll cholesterol load will drop and this is a supposed benefit of this diet that is apparently born out by evidence. Not that the ratios do not seem to alter, just the TC level. This means that any lipids that do escape the liver will not be easily replaced since there are few in the pipeline. BUT cholesterol has many uses. One use is to build and repair cells, so reducing the TC has been shown by science to lead to higher mortality since damaged cells do not get replaced. It may please the doctor but is not as healthy as has been made out.</p><p></p><p>The other thing that a low TC can lead to is muscle wasting. The body need lipid fats for the protein and amino acids to build muscle tissue. Diets that reduce fat too much can force the body to scavenge muscle tissue to get the cholesterol it needs to (a) make new cholesterol, and (b) repair cells using glycerol from dead cholesterol or scavenged from muscle cells. This phenomenon is understood especially by those doing athletics and fitness training and is not specific to this diet, but to any ULF diet.</p><p></p><p>So where this diet claims to remove lipid fat in a short space of time, I am left puzzled. The average T2D with insulin resistance does not give up adipose fat or metabolic syndrome without a fight, It is stubborn to shift at the best of times. We see this diet reduces lipid traffic in the blood, so there is no evidence there that liver lipids are coming out of the liver. so where is it going? Metabolic rate is controlled by the thyroid gland, so is the chemical(C) interfering with its function? Is that safe? Does anyone know? Do the Founders know? What does Chemical(C) do? Does it pop the fat bubbles in storage? Surely the lipds go somewhere, and they cannot go unmasked in the blood (water insoluble) so could be ending up as damaged cholesterol (i.e. trigs) and so present an increased risk of blood clots. This is where the diet plan falls down, It has no explanation of what happens to these lipids. A spare tyre full of blubbering lipids holds an awful lot of energy, so if my metabolism increased then it will have very significant visual clues (overheating, boundless energy, restlessness, bulging eyes, overstimulated thought processes, sleeplessness) </p><p></p><p>I do not buy into their story that chemical(C) is a magic bullet. If it exists, then there will be a pill for it.</p><p></p><p>Anybody got a different theory?</p></blockquote><p></p>
[QUOTE="Oldvatr, post: 2306980, member: 196898"] If I had evidence of, or suspected tht, by taking a chemical from Food(A) and mixing it with a chemical from Food(B) to make a new chemical (C) that had the power to remove adipose fat without disturbing the rest of the lymphatic system, then I would keep stumm about it until I had isolated the process, managed to synthesise it in a testube, make sure I could scale up to making large vats of it in a factory. Then I would make sure I patented it, This has got to be the Golden Goose of this century, and I would not faff about selling diet plans and leaving it open to others to make their fortunes from. Is this why explanations in the website are so poor and insubstantial? I am having real difficulty seeing how this diet would lead to the claims being made for it especially for T2 diabetics. It is unrestricted calories, and one can eat as much as one wants to. It allows foods such as oatmeal, brown rice, quinoa, and bread It is certainly not Keto, nor VLCD, but is very, very low fat. The high level of carbs recommended in unlimited amounts and uncontrolled portion sizes is an anathema to most T2D reading this and it defies both science (carbs directly lead to increasing glucose levels) and personal experiences, and anecdotal evidence throughout this Forum. Even normal non diabetics have a rise in glucose levels when eating carbs. The body is designed to respond to the amylase rushes to give two insulin responses (but T2D have an impaired reaction) and this is how it has been since man first walked the earth. So my first stake in the ground is that a high carb diet will result in a rise in bgl. It is nothing to do with omnivore vs plant based, but is due to diets with high carb intake. Moving on and assuming this stake remains standing, then a T2D eating some foods as recommended will see their bgl start to tise after the meal. Since one hallmark of T2D is insulin resistancem then this will mean that the body has difficulty in either using up or storing the glucose in the blood, and it will tend to remain atelevted levels for some time. This level of bgl is enough to prevent not just storage but also of using stored glucose until bgl levels drop to normal. Importantly it stops the body creating glucose from lipids or emptying the existing glucose stores in the liver. So it keeps what you already got. Adipose fat remains untouched. Gradually bgl levels drop as we expend energy. Since the diet states that exercise is not a requirement of the diet plan then this may take a while. Now considering the adipose fat (tiny lipid particles as the diet states) then this only gets used when the body is not in storage mode, so having high levels of insulin and high glucose levels prevents the body from using lipids as a source of energy. So the average T2D will spend most of the day trying to force excess glucose into storage, which is the hallmark of our condition. But this diet has one thing in its favour. It is ultra low fat (nuts anybody?). This means that the overll cholesterol load will drop and this is a supposed benefit of this diet that is apparently born out by evidence. Not that the ratios do not seem to alter, just the TC level. This means that any lipids that do escape the liver will not be easily replaced since there are few in the pipeline. BUT cholesterol has many uses. One use is to build and repair cells, so reducing the TC has been shown by science to lead to higher mortality since damaged cells do not get replaced. It may please the doctor but is not as healthy as has been made out. The other thing that a low TC can lead to is muscle wasting. The body need lipid fats for the protein and amino acids to build muscle tissue. Diets that reduce fat too much can force the body to scavenge muscle tissue to get the cholesterol it needs to (a) make new cholesterol, and (b) repair cells using glycerol from dead cholesterol or scavenged from muscle cells. This phenomenon is understood especially by those doing athletics and fitness training and is not specific to this diet, but to any ULF diet. So where this diet claims to remove lipid fat in a short space of time, I am left puzzled. The average T2D with insulin resistance does not give up adipose fat or metabolic syndrome without a fight, It is stubborn to shift at the best of times. We see this diet reduces lipid traffic in the blood, so there is no evidence there that liver lipids are coming out of the liver. so where is it going? Metabolic rate is controlled by the thyroid gland, so is the chemical(C) interfering with its function? Is that safe? Does anyone know? Do the Founders know? What does Chemical(C) do? Does it pop the fat bubbles in storage? Surely the lipds go somewhere, and they cannot go unmasked in the blood (water insoluble) so could be ending up as damaged cholesterol (i.e. trigs) and so present an increased risk of blood clots. This is where the diet plan falls down, It has no explanation of what happens to these lipids. A spare tyre full of blubbering lipids holds an awful lot of energy, so if my metabolism increased then it will have very significant visual clues (overheating, boundless energy, restlessness, bulging eyes, overstimulated thought processes, sleeplessness) I do not buy into their story that chemical(C) is a magic bullet. If it exists, then there will be a pill for it. Anybody got a different theory? [/QUOTE]
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