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No test strips or lancets on the NHS?!!!
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<blockquote data-quote="Yorksman" data-source="post: 376709" data-attributes="member: 55568"><p>There are large numbers who are classified as non obese who get it, particularly in asian populations. Ironically, they are less likely to know that they are at risk, because they tend to be thin. The problem is the intra organ fat and, according to Taylor, the liver and pancreas triglycerides. In the west, most obese people do not have diabetes but they know they are at risk because they can see for themselves, fats are accumulating. People like Ran Fiennes who are thin and run marathons don't know the fats are accumulating in their organs. I suspect that types of food together with several interacting genetic mechanisms are involved. In obese patients, the tipping point, the point at which insulin production is adversely affected, is unpredictable. For some patients it may be a BMI of 25 whilst others with a BMI of 35 are unaffected. In non obese patients, the picture is even less clear, but there are large numbers out there.</p><p></p><p>Non-obese patients with type 2 diabetes and prediabetic subjects: distinct phenotypes requiring special diabetes treatment and (or) prevention? <a href="http://www.ncbi.nlm.nih.gov/pubmed/18059616" target="_blank">http://www.ncbi.nlm.nih.gov/pubmed/18059616</a></p><p></p><p>It seems that now the genetics is better understood, type 2 diabetes is starting to be classified as an autoimmune disorder rather than a metabolic disorder.</p><p></p><p>Type-2 diabetes linked to autoimmune reaction in study <a href="http://med.stanford.edu/ism/2011/april/engleman.html" target="_blank">http://med.stanford.edu/ism/2011/april/engleman.html</a></p></blockquote><p></p>
[QUOTE="Yorksman, post: 376709, member: 55568"] There are large numbers who are classified as non obese who get it, particularly in asian populations. Ironically, they are less likely to know that they are at risk, because they tend to be thin. The problem is the intra organ fat and, according to Taylor, the liver and pancreas triglycerides. In the west, most obese people do not have diabetes but they know they are at risk because they can see for themselves, fats are accumulating. People like Ran Fiennes who are thin and run marathons don't know the fats are accumulating in their organs. I suspect that types of food together with several interacting genetic mechanisms are involved. In obese patients, the tipping point, the point at which insulin production is adversely affected, is unpredictable. For some patients it may be a BMI of 25 whilst others with a BMI of 35 are unaffected. In non obese patients, the picture is even less clear, but there are large numbers out there. Non-obese patients with type 2 diabetes and prediabetic subjects: distinct phenotypes requiring special diabetes treatment and (or) prevention? [url=http://www.ncbi.nlm.nih.gov/pubmed/18059616]http://www.ncbi.nlm.nih.gov/pubmed/18059616[/url] It seems that now the genetics is better understood, type 2 diabetes is starting to be classified as an autoimmune disorder rather than a metabolic disorder. Type-2 diabetes linked to autoimmune reaction in study [url=http://med.stanford.edu/ism/2011/april/engleman.html]http://med.stanford.edu/ism/2011/april/engleman.html[/url] [/QUOTE]
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No test strips or lancets on the NHS?!!!
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