Reply to Thread

Message

<blockquote data-quote="NewdestinyX" data-source="post: 395755" data-attributes="member: 35544">Agreed to the &#039;one size fits all&#039; approach letting us ALL down. If Type 2 Diabetes were an &#039;easy sum up&#039;.. Medical science would be able to cure it. They&#039;re having so much trouble finding out the &#039;triggers&#039; of the disease. I have to agree with Sid Bonkers here that a &#039;better lifestyle&#039; is just plain &#039;good&#039; - for ALL. Visceral (around our bellies) fat increases risk for insulin resistance which itself can be a trigger or, to use another poster&#039;s words, &#039;the environment for a gene to express itself&#039;. I am one of those &#039;highly insulin resistant in the presence of visceral fat&#039; guys. I&#039;ve been up and down with the weight over the last 4 years since DX.. and when I&#039;m down I need no long acting insulin and can control with only diet. When I&#039;m up in weight I need a lot more insulin and daily diet has little affect on my numbers. If I got down to my &#039;prescribed weight&#039; I&#039;m convinced I would have NO expression of Diabetes at all. But that&#039;s me. To show how confusing this topic is and why I also AGREE with another poster who said we need new &#039;designations&#039; for sub divisions of Type 2 - the stats are actually this:1) Only 20% of the world&#039;s obese people have or will ever get T2 Diabetes. 2) 80% of the world&#039;s T2 diabetics are obese..Now -- how do you make those two &#039;stats&#039; jibe??? That&#039;s a mind-boggling set of stats and again points to why it&#039;s so hard to classify this disease and paint with broad brush strokes. All I know - is that for me -- ever stone I lose makes controlling my blood sugar easier - to the point where it would be totally &#039;in remission&#039;, if you will, if I were to be &#039;normal weight&#039;.</blockquote>

[QUOTE="NewdestinyX, post: 395755, member: 35544"] Agreed to the 'one size fits all' approach letting us ALL down. If Type 2 Diabetes were an 'easy sum up'.. Medical science would be able to cure it. They're having so much trouble finding out the 'triggers' of the disease. I have to agree with Sid Bonkers here that a 'better lifestyle' is just plain 'good' - for ALL. Visceral (around our bellies) fat increases risk for insulin resistance which itself can be a trigger or, to use another poster's words, 'the environment for a gene to express itself'. I am one of those 'highly insulin resistant in the presence of visceral fat' guys. I've been up and down with the weight over the last 4 years since DX.. and when I'm down I need no long acting insulin and can control with only diet. When I'm up in weight I need a lot more insulin and daily diet has little affect on my numbers. If I got down to my 'prescribed weight' I'm convinced I would have NO expression of Diabetes at all. But that's me. To show how confusing this topic is and why I also AGREE with another poster who said we need new 'designations' for sub divisions of Type 2 - the stats are actually this: 1) Only 20% of the world's obese people have or will ever get T2 Diabetes. 2) 80% of the world's T2 diabetics are obese.. Now -- how do you make those two 'stats' jibe??? That's a mind-boggling set of stats and again points to why it's so hard to classify this disease and paint with broad brush strokes. All I know - is that for me -- ever stone I lose makes controlling my blood sugar easier - to the point where it would be totally 'in remission', if you will, if I were to be 'normal weight'. [/QUOTE]

Verification

Search

Search

Reply to Thread