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Phase 1 insulin response
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<blockquote data-quote="Oldvatr" data-source="post: 2426977" data-attributes="member: 196898"><p>That report reads like it is a thesis or application paper for professorship. I do not think it is a working document, and is not documenting any experimental evidence. It is a super-blog so is somebody's explanation, as a theory. The whole document reads like it is out to baffle the reader and impress the academic body. </p><p>For example:</p><p>"In the postprandial state, a variety of nutritional factors in circulation including amino acids, fatty acids and glucose serve as insulin secretagogues resulting in the release of insulin that initiates signaling cascades responsible for the suppression hepatic glucose output,"</p><p>This is a high faluting way of saying insulin is secreted to lower blood glucose and is triggered by food intake.</p><p></p><p>I would not hang my hat on it yet until someone does some experimental work to validate the theory.</p></blockquote><p></p>
[QUOTE="Oldvatr, post: 2426977, member: 196898"] That report reads like it is a thesis or application paper for professorship. I do not think it is a working document, and is not documenting any experimental evidence. It is a super-blog so is somebody's explanation, as a theory. The whole document reads like it is out to baffle the reader and impress the academic body. For example: "In the postprandial state, a variety of nutritional factors in circulation including amino acids, fatty acids and glucose serve as insulin secretagogues resulting in the release of insulin that initiates signaling cascades responsible for the suppression hepatic glucose output," This is a high faluting way of saying insulin is secreted to lower blood glucose and is triggered by food intake. I would not hang my hat on it yet until someone does some experimental work to validate the theory. [/QUOTE]
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