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<blockquote data-quote="Snodger" data-source="post: 323699" data-attributes="member: 27045"><p>yes, sorry if I came across a bit cold. I think it's totally fine to explore alternatives. That's how we find out useful things. </p><p></p><p></p><p>I'm not an expert but yes, I think it's relatively clear-cut. The healthy body is pretty amazing. Of course, the fat itself would then start to cause problems. In some people (although still not everyone) that extra fat would then start to contribute to insulin resistance (body cells not recognising the insulin very well and so not responding to it)</p><p></p><p>You're quite right to speculate that there COULD be a disease in which the liver got overloaded with glycogen and stopped working, but for some reason human bodies don't seem to do that. The problems seem to focus on insulin production and reception/recognition. </p><p></p><p></p><p>I wouldn't say that it was invariably progressive at all. Certainly it's not with Type 1: we conk out and that's it, we can't make insulin. We don't get progressively worse at making insulin, because we don't make any at all. (Some of us develop insulin resistance as well, but that's slightly different). But I would also say it's not invariably progressive with Type 2 either. Some people do control it with diet for the rest of their lives. You can call that a form of 'diabetes' or you can call that 'impaired glucose intolerance' but either way it's not the same as the healthy person whose body responds efficiently to whatever foods they eat. You won't get that ability back again, whatever you do, so as far as that goes you are permanently diabetic - even if you are able to keep off medication.</p></blockquote><p></p>
[QUOTE="Snodger, post: 323699, member: 27045"] yes, sorry if I came across a bit cold. I think it's totally fine to explore alternatives. That's how we find out useful things. I'm not an expert but yes, I think it's relatively clear-cut. The healthy body is pretty amazing. Of course, the fat itself would then start to cause problems. In some people (although still not everyone) that extra fat would then start to contribute to insulin resistance (body cells not recognising the insulin very well and so not responding to it) You're quite right to speculate that there COULD be a disease in which the liver got overloaded with glycogen and stopped working, but for some reason human bodies don't seem to do that. The problems seem to focus on insulin production and reception/recognition. I wouldn't say that it was invariably progressive at all. Certainly it's not with Type 1: we conk out and that's it, we can't make insulin. We don't get progressively worse at making insulin, because we don't make any at all. (Some of us develop insulin resistance as well, but that's slightly different). But I would also say it's not invariably progressive with Type 2 either. Some people do control it with diet for the rest of their lives. You can call that a form of 'diabetes' or you can call that 'impaired glucose intolerance' but either way it's not the same as the healthy person whose body responds efficiently to whatever foods they eat. You won't get that ability back again, whatever you do, so as far as that goes you are permanently diabetic - even if you are able to keep off medication. [/QUOTE]
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