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<blockquote data-quote="Bluetit1802" data-source="post: 1581101" data-attributes="member: 94045"><p>It is all part of how our bodies work naturally. The pancreas and the liver work in tandem, regulated by hormones.</p><p></p><p>In normal healthy people the liver will dump glucose into the blood stream from its own stores whenever the hormones tell it the body is low on glucose for its immediate needs, such as overnight fasting, between meals etc. The pancreas picks up on this and secretes insulin to get to work pushing this glucose into the cells that need it. Job done. Glucose provided by the liver and carried by the insulin to where it is needed. The hormones then tell the liver to stop dumping (until the next time). We don't die. We don't hypo. No-one has zero glucose. It is a natural event designed to keep us alive.</p><p></p><p>In diabetics, this system breaks down. In T2s this is either because the pancreas cannot produce enough insulin, or because the insulin it produces is rejected by the cells due to insulin resistance. (Sometimes the reason can also be wonky hormones not giving out the right messages.) So we end up with too much circulating glucose (and insulin)</p><p></p><p>I have no idea what happens in the case of T1s but imagine it can be a nightmare situation for them having to juggle the injected insulin at the right time to clear the liver dumps.</p><p></p><p>I am dashing right now, but will try to find some links later.</p></blockquote><p></p>
[QUOTE="Bluetit1802, post: 1581101, member: 94045"] It is all part of how our bodies work naturally. The pancreas and the liver work in tandem, regulated by hormones. In normal healthy people the liver will dump glucose into the blood stream from its own stores whenever the hormones tell it the body is low on glucose for its immediate needs, such as overnight fasting, between meals etc. The pancreas picks up on this and secretes insulin to get to work pushing this glucose into the cells that need it. Job done. Glucose provided by the liver and carried by the insulin to where it is needed. The hormones then tell the liver to stop dumping (until the next time). We don't die. We don't hypo. No-one has zero glucose. It is a natural event designed to keep us alive. In diabetics, this system breaks down. In T2s this is either because the pancreas cannot produce enough insulin, or because the insulin it produces is rejected by the cells due to insulin resistance. (Sometimes the reason can also be wonky hormones not giving out the right messages.) So we end up with too much circulating glucose (and insulin) I have no idea what happens in the case of T1s but imagine it can be a nightmare situation for them having to juggle the injected insulin at the right time to clear the liver dumps. I am dashing right now, but will try to find some links later. [/QUOTE]
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