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Reverse Type 2 Diabetes with a LCHF diet. Is this a myth?
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<blockquote data-quote="NicoleC1971" data-source="post: 2174445" data-attributes="member: 365308"><p>Hi and welcome. Great questions and I doubt a doctor will bother having this discussion with you. I always want to know how my type 1 diabetes work but the whole debate on diet is fascinating though apt to be partisan at times when keto or carnivores/the Ruminati discuss this with vegans! Nutritional science is young and notoriously hard to prove a hypothesis in.</p><p>My take on the human diet is that whilst we evolved to be omnivorous but eating meat enabled us to grow big brains eventually leading to us taming wild grasses into crops, then becoming 'civillised' and eventually inventing foodstuffs that we mainly fail to thrive on. </p><p>The amount of grains would have depended on your local agriculture thus some humans have more starch processing enzymes (amalayse) than others and many cultures have thrived by eating large quantities of starch whereas the converse is true for others (Aborigines and Inuits) thus there is a variance in how much starch we can tolerate before it gets stored in the liver and around the pancreas thus leaving to metabolic problems such as type 2.</p><p>On the other hand, the most recent part of our history involves eating large amounts of processed carbs and sugars at the expense of nutrient dense whole foods including animal fats due to the parallel fear of saturated fat leading to CVD (a failed hypothesis). Many people have causally linked this change in our diet to the rise of diabobesity (see Robert Lustig on Sugar). The contention of such people as Lustig or Jason fung is that obesity does not cause diabetes but is a downstream effect of a hormonal imbalance driven by excess insulin with excess insulin patterns driven by excess sugar (a fructose overload that the liver cannot handle without fattening) and/or processed carbs needing to be stored as fat by insulin until the person's individual fat storage capacity is reached. Some people can store lots of sub cutaneous fat but don't become diabetic and this is perhaps why. Others are surprised to be diagnosed when still slim (e.g. Michael Mosley). </p><p>So any way of eating that rapidly strips down excess stored liver fat by minimising insulin requirement will work to increase someone's insulin sensitivity (as will building more muscle). this could be the Newcastle protocol, fasting or low carb/high fat and the more common commercial diets could still work (i.e. low fat, moderate carb) depending on where the person is starting from because all diets restrict carbs to some extent. However many people feel that low carb/high fat is the best at being sustainable as a lifestyle rather than a quick fix followed by difficulty in establishing an eating pattern that doesn't result in rebounding back into weight gain and/or high blood sugars. the latter is anecdotal but if you go to Public Health Consortium's site you will find comparison of the dietary trials comparing low carb with low fat diets etc.</p><p>As for the term reversal vs remission, that's semantic IMO! The work is in recovering normal insulin glucose levels and then finding a way to keep them normal. And there's nothing as powerful as diet and lifestyle for that but some people will need more time and drugs or even bariatric surgery based on individual factors such as level of IR and its duration, pancreatic function or simply an addiction to carbs!</p></blockquote><p></p>
[QUOTE="NicoleC1971, post: 2174445, member: 365308"] Hi and welcome. Great questions and I doubt a doctor will bother having this discussion with you. I always want to know how my type 1 diabetes work but the whole debate on diet is fascinating though apt to be partisan at times when keto or carnivores/the Ruminati discuss this with vegans! Nutritional science is young and notoriously hard to prove a hypothesis in. My take on the human diet is that whilst we evolved to be omnivorous but eating meat enabled us to grow big brains eventually leading to us taming wild grasses into crops, then becoming 'civillised' and eventually inventing foodstuffs that we mainly fail to thrive on. The amount of grains would have depended on your local agriculture thus some humans have more starch processing enzymes (amalayse) than others and many cultures have thrived by eating large quantities of starch whereas the converse is true for others (Aborigines and Inuits) thus there is a variance in how much starch we can tolerate before it gets stored in the liver and around the pancreas thus leaving to metabolic problems such as type 2. On the other hand, the most recent part of our history involves eating large amounts of processed carbs and sugars at the expense of nutrient dense whole foods including animal fats due to the parallel fear of saturated fat leading to CVD (a failed hypothesis). Many people have causally linked this change in our diet to the rise of diabobesity (see Robert Lustig on Sugar). The contention of such people as Lustig or Jason fung is that obesity does not cause diabetes but is a downstream effect of a hormonal imbalance driven by excess insulin with excess insulin patterns driven by excess sugar (a fructose overload that the liver cannot handle without fattening) and/or processed carbs needing to be stored as fat by insulin until the person's individual fat storage capacity is reached. Some people can store lots of sub cutaneous fat but don't become diabetic and this is perhaps why. Others are surprised to be diagnosed when still slim (e.g. Michael Mosley). So any way of eating that rapidly strips down excess stored liver fat by minimising insulin requirement will work to increase someone's insulin sensitivity (as will building more muscle). this could be the Newcastle protocol, fasting or low carb/high fat and the more common commercial diets could still work (i.e. low fat, moderate carb) depending on where the person is starting from because all diets restrict carbs to some extent. However many people feel that low carb/high fat is the best at being sustainable as a lifestyle rather than a quick fix followed by difficulty in establishing an eating pattern that doesn't result in rebounding back into weight gain and/or high blood sugars. the latter is anecdotal but if you go to Public Health Consortium's site you will find comparison of the dietary trials comparing low carb with low fat diets etc. As for the term reversal vs remission, that's semantic IMO! The work is in recovering normal insulin glucose levels and then finding a way to keep them normal. And there's nothing as powerful as diet and lifestyle for that but some people will need more time and drugs or even bariatric surgery based on individual factors such as level of IR and its duration, pancreatic function or simply an addiction to carbs! [/QUOTE]
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