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Type 1 Diabetic - Obese and addicted to food
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<blockquote data-quote="Ellenor2000" data-source="post: 2227033" data-attributes="member: 508208"><p>Because they want to do anything except believe that the pathology of hyperadiposity is caused by chronic overdosing of insulin (carb-fat diets, low fat in the susceptible also, cortisol, and fructose abuse) and that alone. Even the "leptin resistance" (in reality, it's another signal that overrides the high leptin and makes you eat even though you aren't hungry) is downstream of the insulin. (OP of course needs to take insulin if they do not want to suffer from a fatal catabolic disease along with high blood sugars - I am not saying insulin is bad, only that overdosing insulin, even in the absence of resulting hypoglycemia, is bad.)</p><p></p><p>Obesity research is a "scientific" field full of non-science. They're basically chasing cold fusion up in that thing.</p><p></p><p>If leptin resistance (as in, a reduction in response to leptin as levels rise independently of any other factor) was real, you would expect fat people to have symptoms of hypothyroidism (since low leptin causes a low triiodothyronine level), indistinguishable from someone who was fat because they actually were hypothyroid. That's not happening.</p></blockquote><p></p>
[QUOTE="Ellenor2000, post: 2227033, member: 508208"] Because they want to do anything except believe that the pathology of hyperadiposity is caused by chronic overdosing of insulin (carb-fat diets, low fat in the susceptible also, cortisol, and fructose abuse) and that alone. Even the "leptin resistance" (in reality, it's another signal that overrides the high leptin and makes you eat even though you aren't hungry) is downstream of the insulin. (OP of course needs to take insulin if they do not want to suffer from a fatal catabolic disease along with high blood sugars - I am not saying insulin is bad, only that overdosing insulin, even in the absence of resulting hypoglycemia, is bad.) Obesity research is a "scientific" field full of non-science. They're basically chasing cold fusion up in that thing. If leptin resistance (as in, a reduction in response to leptin as levels rise independently of any other factor) was real, you would expect fat people to have symptoms of hypothyroidism (since low leptin causes a low triiodothyronine level), indistinguishable from someone who was fat because they actually were hypothyroid. That's not happening. [/QUOTE]
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