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Type 3c (Pancreatic) Diabetes
Type 3c, taking insulin, good/bad?
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<blockquote data-quote="Civilised Devil" data-source="post: 2388213" data-attributes="member: 524083"><p>Hello and thank you for your reply, I,m still reading up on 3c there is a lot of good informative articles out there. The one constant that keeps appearing is the common misdiagnosis and misunderstanding of 3c and is often diagnosed as T2. 3c is the pancreas inability to make insulin because of damage done to the beta cells eg as in pancreatitis, as far as I can tell this is more similar to T 1 were the Insulin making beta cells are attacked and destroyed by the body's immune system. So both are not making Insulin.</p><p> Insulin resistance (IR) is a pathological condition in which cells fail to respond normally to the hormone insulin. How is this relevant to 3c? I do not mean to come across as flippant but Insulin resistance is more a T2 condition, no? Type 3c is about not producing insulin in the first place.</p><p>Have scanned Jason tung's blog and your right it is aimed at T2 an so not really relevant, for a lot of pancreatitis sufferers fat is the enemy, of course the body needs fat but even with enzyme replacement therapy it can trigger an attack so a more holistic approach is needed that addresses the needs of diabetes and pancreatitis togeather.</p><p>Speculation is the first step to a hypothesis and is always welcome and you may be right every attack might destroy more beta cells. The lag in time between attack and need for insulin is what concerns me, that and the insulin needed has increased as I increase the dose, hence the relevance of the bodybuilders story. Speculation yes, but it's about considering what might be possible based on observation and factual information so the question is legitimate, the bodybuilders made themselves diabetic by replacing the function of their pancreas, the beta cells stopped working.</p><p>I didn't know the pancreas can take a rest from insulin production, how is that triggered? What conditions are necessary for that to happen? And why? How does it restart? What are the conditions for it to restart? can you post a link to the relevant papers that would be most interesting. My own situation is that when the pancreas is inflamed production stops but slowly returns as the pancreas gets de-inflammed, returns to normal as does the insulin levels it has been fairly long time since my last big attack BG levels were normal then lately were slightly high but have increased significantly since the reintroduction of Lantus and now with novorapid.</p><p>The most difficult thing is getting control of the diabetes, just when you think you've got the dose right, pancreas decides to start working and BG readings of 3.0 and less from what I have read this is fairly common, wild swings in BG. my post and questions are about finding reasons and understanding for this.</p><p>Stay well.</p></blockquote><p></p>
[QUOTE="Civilised Devil, post: 2388213, member: 524083"] Hello and thank you for your reply, I,m still reading up on 3c there is a lot of good informative articles out there. The one constant that keeps appearing is the common misdiagnosis and misunderstanding of 3c and is often diagnosed as T2. 3c is the pancreas inability to make insulin because of damage done to the beta cells eg as in pancreatitis, as far as I can tell this is more similar to T 1 were the Insulin making beta cells are attacked and destroyed by the body's immune system. So both are not making Insulin. Insulin resistance (IR) is a pathological condition in which cells fail to respond normally to the hormone insulin. How is this relevant to 3c? I do not mean to come across as flippant but Insulin resistance is more a T2 condition, no? Type 3c is about not producing insulin in the first place. Have scanned Jason tung's blog and your right it is aimed at T2 an so not really relevant, for a lot of pancreatitis sufferers fat is the enemy, of course the body needs fat but even with enzyme replacement therapy it can trigger an attack so a more holistic approach is needed that addresses the needs of diabetes and pancreatitis togeather. Speculation is the first step to a hypothesis and is always welcome and you may be right every attack might destroy more beta cells. The lag in time between attack and need for insulin is what concerns me, that and the insulin needed has increased as I increase the dose, hence the relevance of the bodybuilders story. Speculation yes, but it's about considering what might be possible based on observation and factual information so the question is legitimate, the bodybuilders made themselves diabetic by replacing the function of their pancreas, the beta cells stopped working. I didn't know the pancreas can take a rest from insulin production, how is that triggered? What conditions are necessary for that to happen? And why? How does it restart? What are the conditions for it to restart? can you post a link to the relevant papers that would be most interesting. My own situation is that when the pancreas is inflamed production stops but slowly returns as the pancreas gets de-inflammed, returns to normal as does the insulin levels it has been fairly long time since my last big attack BG levels were normal then lately were slightly high but have increased significantly since the reintroduction of Lantus and now with novorapid. The most difficult thing is getting control of the diabetes, just when you think you've got the dose right, pancreas decides to start working and BG readings of 3.0 and less from what I have read this is fairly common, wild swings in BG. my post and questions are about finding reasons and understanding for this. Stay well. [/QUOTE]
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