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Type 3c (Pancreatic) Diabetes
Type 3c, taking insulin, good/bad?
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<blockquote data-quote="Brunneria" data-source="post: 2388231" data-attributes="member: 41816"><p>Anyone can experience insulin resistance. All it needs is enough insulin floating about for the body to become resistant. It can happen for a number of reasons, including genetics, lifestyle, being obese or thin outside, fat inside (visceral fat), certain medications, NAFLD, certain hormonal disruptions, and other reasons. It certainly isn't restricted to T2s, although it is most common to them. This is why I recommended Jason Fung's blog, because he goes into excellent detail describing the whys, wherefores, and solutions. You (potentially) could have both 3c and insulin resistance. I think it is certainly more likely than some mythical shared experience with body builders.</p><p></p><p>Lots of T2s get prescribed insulin at diagnosis, but once their system settles, and they sort out lifestyle and diet, they may be able to significantly reduce their medication, or remove meds altogether. Also, for T1s and LADAs, there is something called the honeymoon period, where (after diagnosis) their pancreas seems to recover a bit, and they may be able to reduce or remove insulin for a while, although with T1 insulin will become a necessity eventually. You would have to ask your consultant whether 3cers get a honeymoon.</p><p></p><p>Your description of the difficulty of dosing when the insulin production increases and decreases unpredictably sounds like classic honeymoon to me. But I expect that no one, even your consultant, can predict whether you will continue to lose beta cells, or not.</p><p></p><p>Taking exogenous insulin doesn't kill beta cells. Sustained high blood glucose kills beta cells. So (for T1s) does the body's own immune system. Also, having a fatty liver can effectively 'smother' beta cells (which have recently been shown Professor Taylor's Newcastle Diet studies to return to function once the fat has been removed - for a minority of those undergoing the weight loss in the studies). And then, of course, pancreatitis can kill beta cells too.</p></blockquote><p></p>
[QUOTE="Brunneria, post: 2388231, member: 41816"] Anyone can experience insulin resistance. All it needs is enough insulin floating about for the body to become resistant. It can happen for a number of reasons, including genetics, lifestyle, being obese or thin outside, fat inside (visceral fat), certain medications, NAFLD, certain hormonal disruptions, and other reasons. It certainly isn't restricted to T2s, although it is most common to them. This is why I recommended Jason Fung's blog, because he goes into excellent detail describing the whys, wherefores, and solutions. You (potentially) could have both 3c and insulin resistance. I think it is certainly more likely than some mythical shared experience with body builders. Lots of T2s get prescribed insulin at diagnosis, but once their system settles, and they sort out lifestyle and diet, they may be able to significantly reduce their medication, or remove meds altogether. Also, for T1s and LADAs, there is something called the honeymoon period, where (after diagnosis) their pancreas seems to recover a bit, and they may be able to reduce or remove insulin for a while, although with T1 insulin will become a necessity eventually. You would have to ask your consultant whether 3cers get a honeymoon. Your description of the difficulty of dosing when the insulin production increases and decreases unpredictably sounds like classic honeymoon to me. But I expect that no one, even your consultant, can predict whether you will continue to lose beta cells, or not. Taking exogenous insulin doesn't kill beta cells. Sustained high blood glucose kills beta cells. So (for T1s) does the body's own immune system. Also, having a fatty liver can effectively 'smother' beta cells (which have recently been shown Professor Taylor's Newcastle Diet studies to return to function once the fat has been removed - for a minority of those undergoing the weight loss in the studies). And then, of course, pancreatitis can kill beta cells too. [/QUOTE]
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