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Undiagnosed but high blood sugar
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<blockquote data-quote="Brunneria" data-source="post: 869518" data-attributes="member: 41816"><p>Hi [USER=36236]@BaliRob[/USER]</p><p></p><p>I'm afraid you will find that confusion is obligatory. <img src="data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7" class="smilie smilie--sprite smilie--sprite2" alt=";)" title="Wink ;)" loading="lazy" data-shortname=";)" /></p><p></p><p>Type 2 is a highly complex thing, and we all have a subtly different way that it works in our bodies.</p><p></p><p>When you factor in 1st and 2nd insulin responses, stress, exercise, additional hormone reactions, insulin resistance, age, beta cell deterioration, personal fat thresholds and personal carb tolerances, as well as what we choose to eat, the permutations really are infinite.</p><p></p><p>Me? I started out with reactive hypoglycaemia, which lasted for about 30 yrs of excessive insulin production, reactive hypoglycaemic lows, accompanying weight and insulin resistance. Plus other hormone nonsense. Eventually my poor overused beta cells began to give out, and I now have a fun combo of RH and T2.</p><p></p><p>As far as I can tell, I have virtually no 1st insulin response, and my second response is sluggish but excessive. Either I produce too much insulin, too late, or I release too little of glucagon (the hormone that 'turns insulin off') leading to those reactive hypos.</p><p></p><p>In contrast, most type 2s never experience hypos - so their issues are very different.</p><p></p><p>Sometimes they produce enough insulin, but it is delayed, leading to damagingly high BG until the insulin turns up. Or their beta cells are clogged with fat, so they can't work properly.</p><p></p><p>But we all fit under the T2 umbrella.</p><p>The trick is to find out what works for you - then stick to it. <img src="data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7" class="smilie smilie--sprite smilie--sprite1" alt=":)" title="Smile :)" loading="lazy" data-shortname=":)" /></p></blockquote><p></p>
[QUOTE="Brunneria, post: 869518, member: 41816"] Hi [USER=36236]@BaliRob[/USER] I'm afraid you will find that confusion is obligatory. ;) Type 2 is a highly complex thing, and we all have a subtly different way that it works in our bodies. When you factor in 1st and 2nd insulin responses, stress, exercise, additional hormone reactions, insulin resistance, age, beta cell deterioration, personal fat thresholds and personal carb tolerances, as well as what we choose to eat, the permutations really are infinite. Me? I started out with reactive hypoglycaemia, which lasted for about 30 yrs of excessive insulin production, reactive hypoglycaemic lows, accompanying weight and insulin resistance. Plus other hormone nonsense. Eventually my poor overused beta cells began to give out, and I now have a fun combo of RH and T2. As far as I can tell, I have virtually no 1st insulin response, and my second response is sluggish but excessive. Either I produce too much insulin, too late, or I release too little of glucagon (the hormone that 'turns insulin off') leading to those reactive hypos. In contrast, most type 2s never experience hypos - so their issues are very different. Sometimes they produce enough insulin, but it is delayed, leading to damagingly high BG until the insulin turns up. Or their beta cells are clogged with fat, so they can't work properly. But we all fit under the T2 umbrella. The trick is to find out what works for you - then stick to it. :) [/QUOTE]
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