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<blockquote data-quote="ketointheuk" data-source="post: 2288814" data-attributes="member: 469532"><p>Can someone shed some light over this? From what I undersand GLP-1 agonists simply trick our pancreas in producing more insulin and less glucagon... how is that a good thing?</p><p>Type 2 especially comes from hyperinsulinemia anyway... why would be want higher levels of insulin?</p><p>I get that it must knock down glucose and therefore only looking at hba1c that lloks good I guess? But all the data shows how we basically have more of "lower glucose post prandial than fasted" effect which is literally my issue of my entire life: I spout out too much insulin, that kept my glucose and hba1c officially lower and non diabetic but the levels of insulin were off the roof.</p><p>How is getting us to produce more insulin good, how can it possibly makes us lose weight? Help a girl out cause biologically it doesn't make sense to me</p></blockquote><p></p>
[QUOTE="ketointheuk, post: 2288814, member: 469532"] Can someone shed some light over this? From what I undersand GLP-1 agonists simply trick our pancreas in producing more insulin and less glucagon... how is that a good thing? Type 2 especially comes from hyperinsulinemia anyway... why would be want higher levels of insulin? I get that it must knock down glucose and therefore only looking at hba1c that lloks good I guess? But all the data shows how we basically have more of "lower glucose post prandial than fasted" effect which is literally my issue of my entire life: I spout out too much insulin, that kept my glucose and hba1c officially lower and non diabetic but the levels of insulin were off the roof. How is getting us to produce more insulin good, how can it possibly makes us lose weight? Help a girl out cause biologically it doesn't make sense to me [/QUOTE]
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