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VLCD - Liquid vs Solid
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<blockquote data-quote="Oldvatr" data-source="post: 1659015" data-attributes="member: 196898"><p>When I was diagnosed I was taught that T2D differed from others mainly in having Insulin Resistnce (IR) which was termed mitochondrial IR since it was a failing in the Krebs cycls (i,e, citric cycle) and limited to muscle tissue. There was no definition of adipose IR which is due to NAFLD fat buildup.</p><p></p><p>The one good thing I take from ND is their use of MRI scanning to demonsrate that reduction in adipose fat is directly linked to IR reduction and improved metabolism keeping bgl low even without meds, and that it is a sustainable state (or seems to be for a while at least). The bad thing I get from ND is the interpretation by the press et al that the ND gives remission by reducing IR through weight loss. As far as I can see, adipose IR is the only one being addressed, and the question of mitochondrial IR is forgotten in the excitement.</p><p></p><p>I say that both types of IR need to be removed for remission to be truly in place, amd the jury is still out.</p><p></p><p>So to follow ND by LCHF type diet is sensible since the LC diet will also reduce the need for insulin and this mitigates the effect of mitochondrial IR, but IMHO LCHF has not been shown to cure muscle IR. I also think a keto diet and or intermittent fasting will also reduce adipose IR like ND does (which is itself technically a mild keto diet according to Optifast,)</p><p></p><p>People are desperate to find the magic bullet the quick fix that will remove the drudgery of taking responsibility for one's care. I personally find the challenges a welcome tool in keeping my eye on the ball and my health in as fine a fettle as I can do in the circumstances.</p></blockquote><p></p>
[QUOTE="Oldvatr, post: 1659015, member: 196898"] When I was diagnosed I was taught that T2D differed from others mainly in having Insulin Resistnce (IR) which was termed mitochondrial IR since it was a failing in the Krebs cycls (i,e, citric cycle) and limited to muscle tissue. There was no definition of adipose IR which is due to NAFLD fat buildup. The one good thing I take from ND is their use of MRI scanning to demonsrate that reduction in adipose fat is directly linked to IR reduction and improved metabolism keeping bgl low even without meds, and that it is a sustainable state (or seems to be for a while at least). The bad thing I get from ND is the interpretation by the press et al that the ND gives remission by reducing IR through weight loss. As far as I can see, adipose IR is the only one being addressed, and the question of mitochondrial IR is forgotten in the excitement. I say that both types of IR need to be removed for remission to be truly in place, amd the jury is still out. So to follow ND by LCHF type diet is sensible since the LC diet will also reduce the need for insulin and this mitigates the effect of mitochondrial IR, but IMHO LCHF has not been shown to cure muscle IR. I also think a keto diet and or intermittent fasting will also reduce adipose IR like ND does (which is itself technically a mild keto diet according to Optifast,) People are desperate to find the magic bullet the quick fix that will remove the drudgery of taking responsibility for one's care. I personally find the challenges a welcome tool in keeping my eye on the ball and my health in as fine a fettle as I can do in the circumstances. [/QUOTE]
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