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<blockquote data-quote="Oldvatr" data-source="post: 1555139" data-attributes="member: 196898"><p>I see he disagrees with the notion that LDL can enter the arterial walls, which is plausible since LDL is s large molecule. At the end he says it must be due to endothelial damage, but there is no link to a follow on discussing this, so I am in the dark as to what he reckons causes the plaques. The recent discovery of sLDL which is oxidised or damaged LDL is something he has not taken on board yet. It is much smaller than LDL per se, and is ahown in 2011 as being significant risk factor for artheriosclerosis.</p><p><a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3038964/" target="_blank">https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3038964/</a></p><p></p><p>See also</p><p><a href="https://www.hindawi.com/journals/omcl/2017/1273042/" target="_blank">https://www.hindawi.com/journals/omcl/2017/1273042/</a></p></blockquote><p></p>
[QUOTE="Oldvatr, post: 1555139, member: 196898"] I see he disagrees with the notion that LDL can enter the arterial walls, which is plausible since LDL is s large molecule. At the end he says it must be due to endothelial damage, but there is no link to a follow on discussing this, so I am in the dark as to what he reckons causes the plaques. The recent discovery of sLDL which is oxidised or damaged LDL is something he has not taken on board yet. It is much smaller than LDL per se, and is ahown in 2011 as being significant risk factor for artheriosclerosis. [URL]https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3038964/[/URL] See also [URL]https://www.hindawi.com/journals/omcl/2017/1273042/[/URL] [/QUOTE]
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