I think it probably means as fat increases then the odds become so great it's almost a certainty.
One of the most prevalent theories today is that when fat is safely ensconced in fat cells it doesn't actually cause a problem. This is where fat is supposed to be stored.
Once fat cells are full, they become dysfunctional.. Fat has to find other places, and starts a whole cascade of processes leading to insulin resistance and failing beta cells. People have different fat cell capacities; some have fewer cells, some have small cells, some have more cells and some have larger cells. That's why some people develop T2 at quite low levels of fat (there are even people that have lipodistrophy and don't have many fat cells at all, they are outwardly very thin but have a lot of fat around the organs and are extremely insulin resistant needing hundreds or even thousands of units of insulin)
' Those who don't get diabetes are not filled their fat depots up to the limits and hence have not reached the step that elicit the processes that induce insulin resistance.' (Global Diabetes Course Prof. Thorkild I A Sorensen )
There must be a limit to each persons fat capacity. The 'primary depot' of fat is in the hip and thigh those that are developing a pattern of lots of stomach /trunk fat are more likely to be approaching that limit.
