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The amount of insulin required is a good indication. With LADA there is still a lot of insulin production, at least in the early stages. So the required insulin doses tend to be small. Having said that, it doesn't really matter. Both Type 1 and LADA require the same treatment.

From your first post it sounds like you were diagnosed Type 2. I would want that diagnosis confirmed with an antibody test. If it is in fact Type 1, you would need insulin.

When the new analogue insulin stop work, it is not because of a change in the insulin. It is because of a change in how the body responds to it. In other words, reduced insulin sensitivity. Eating low carb addresses this by removing the need for a quick response to insulin. Exercise has a...

Food containing carbohydrate increases blood glucose. Look at nutritional information on processed food labels to see how much carb it contains. In general terms, the white foods contain lots of carbs. Things like bread, cereal, pasta, potatoes etc. Non starchy vegetables, salads and protein...

That term raises various reasons. What exactly is brittle diabetes? Why don't T1s eating low carb or keto ever have it? What is the difference between poorly controlled and brittle diabetes? And what evidence backs up the assertion that these people don't make glucagon when required?

A doctor in Dunedin murdered his wife using this approach. She was ill but not a diabetic and he injected insulin every day until she died. It took three weeks. A single insulin dose is seldom fatal, though, and attempted suicide using insulin is usually not successful.

The faster an bigger the drop in blood glucose, the stronger the stress response. More adrenalin is secreted, which causes increased anxiety. Slow typos don't do this. It is even possible to sleep through them.

Generally, death where hypos are suspected are often caused by something else. There is no way of telling post-mortem what blood glucose was at the time of death, so when hypoglycemia appears on a death certificate, it is guesswork. With the so-called dead-in-bed syndrome, death is now thought...

Read the second paragraph. The numbers differ somewhat to what at I have seen elsewhere, but in the same ballpark. Note that glucose from muscle glycogen can not get back into the bloodstream, so is no help during a hypo.

The only useful suggestion in this regard is to limit insulin injection amounts to a level that doesn't overwhelm liver glycogen stores. In the average person, the liver holds about 130 grams of glycogen. The similar amount of glycogen in muscles has to be used there and is no use during hypos...

By 'this' are you referring to the idea that the glucagon driven element of the counterregulatory system works for people with T1 diabetes the same as it does for non-diabetics? This is not an outlandish idea. It simply suggests that this element of the metabolism works the way it should. More...

Because insulin is injected subcutaneously and insulin switches Glucagon secretion off, T1s usually have too much of it. Not too little. Glucagon is made in the alpha cells, not the beta cells. There is no reason why a hypo induced stress response wouldn't result in the required glucagon being...

We are all different and blood glucose as measured by HBA1c is not the only factor. For me background retinopathy disappeared when HBA1c fell below about 7% (53mmol/m). I suspect time-in-range, stress etc are also issues.

My response was to the OP, someone taking 25 units of basal a day. So clearly insulin sensitive. There was also clear anxiety about hypos, which is not uncommon. Hypos can happen for various reasons, and reducing the size of insulin shots always reduces this risk. Regardless of total daily dose...

Isn't it odd that we are encouraged to use a practice that increases the risk?

The liver holds enough glucose to deal with most hypos, so you would probably have been ok even if you had slept through it. Having said that, 25 units is a lot of insulin to inject all at once. That doctors are comfortable with this practice puzzles me. Splitting the dose over two shots...

Most peole who have been T1 for 20+ years have had pre-proliferative retinopathy. Also called 'background retinopathy'. There are some microaneurysms, which means there are some blockages. But they are not so bad that new blood vessels, which often burst, have opened up. I had it for years but...

You must be very insulin resistant. The most effective way to improve insulin sensitivity is by getting regular exercise. 30 minutes every day. Metformin may also help. It tends to reduce hunger, reduce liver glucose, and it is also thought to improve insulin sensitivity.

Pre-proliferative retinopathy is when some micro aneurisms can be seen in an eye scan. It is a sign that there are some blockages there but it has not got to the point where new blood vessels are being made. New blood vessels tend to be fragile and burst. This is 'proliferative retinopathy' and...