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Message: <blockquote data-quote="Chris24Main" data-source="post: 2742459" data-attributes="member: 585131">Totally understand what you are saying [USER=588965]@Vectian[/USER] - indeed a lot of what I find myself posting these days revolves around there being a perception that there is a grey area between T1 and T2 - there is not, and the two are opposite in terms of treatment.I guess the only thing I can do is to repeat my story - I was treated for nearly a year as a T1 (late onset or not, fundamentally as you say, T1 is characterised by autoimmune attack on the Pancreas) and given insulin. The effect of that insulin was pretty horrendous, and by the end of that year, my dosage had doubled - apparently confirming that I needed the insulin. However - as soon as I had a C-Peptide test that showed my pancreas was functioning - this was clearly not the case.What I was relating in the post you quoted, was how all of this was explained to me at the time, as I recall being completely confused, - it all seemed to be a question of something going gradually wrong with my pancreas either way, (either way meaning T1/T2) and so I was lacking in insulin, and so adding additional seemed like the right thing to do.I now understand that I had Too Much insulin, which had led to insulin resistance, meaning that the opposite treatment - Lower the Insulin - was appropriate, and so after taking more responsibility for myself, I actually got my bloods under control in a few months and was listed in remission after 3 consecutive HbA1cs.- What I am therefore saying more explicitly is that I had been mis-diagnosed as a T1 when I was not. I cannot of course comment on [USER=582036]@rayq81[/USER] and his diagnosis, but it seems unlikely to me that his Pancreas has suddenly come under auto-immune attack at his age, then agreed some kind of cease-fire for 10 months while he keeps his carbs (therefore insulin) low. It seems much more likely to me, given his C-Peptide is also showing continued function in the β cells, that the underlying issue is insulin resistance, and not a failing pancreas - therefore it seems better for him, given his fantastic efforts; to continue, as opposed to assuming that it's T1 all along, and the auto-immune attack is only waiting to break that cease-fire.on a purely statistical basis - 90% of diabetes is T2 - 8% T1 and ~2% is Late onset T1 - so the odds are in favour of it being insulin resistance, therefore reversible - therefore worth trying to do that.or - it could be that simply reducing carbs lowers chronic inflammation, which allows the body more resource to properly manage the auto-immune system; this is one area of research I need to spend more time on; there is a case that the underlying cause of T1 is also a diet too high in carbs and inflammatory oils - just overwhelming the immune system and causing it to go wrong - leading to lupus in some and an attack on the pancreas in others - but all of that is clearly speculation on my part. I'm just saying that all of this is only my opinion based on my own experience, nothing more. But - rather than trying to figure out what kind of complicated version of diabetes that is LADA, yet still leaves a functioning pancreas, yet at some point will still need insulin - is it not better to really go for remission with diet, as [USER=582036]@rayq81[/USER] is doing (or even yourself)? it strikes me that even if that turns out to be wrong, and it is some extremely rare form of LADA, it's still better for as long as possible.On a side note - you talk about fatigue - do you want to elaborate on that? could you give more information about what in your diet is keeping you insulin- free? (sorry for the clumsy phrase, I just don't want to put words in your mouth...)</blockquote>

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