yetta2mymom
Well-Known Member
- Messages
- 337
- Location
- Winchester Massachusetts
- Type of diabetes
- Don't have diabetes
- Treatment type
- Diet only
- Dislikes
- ?
I live on only protein and a little vegetables I know of people with reactive hypoglycemia who must keep their blood sugar low or they have an insulin reaction and we have problems. So it is possible (maybe not social) to eat such that you never need insulin for making sugar to become fat. I have seen people who just don't believe anyone can eat like I or other people with reactive hypoglycemia do to prevent low blood sugar. I know of support groups which just teach what to do when you have low blood sugar so only a small group of people have the (will?) to eat this way. My type of reactive hypoglycemia is not helped by the support groups since we do not get very low sugar unless we eat a lot of sugar/starch/alcohol we get debilitating fatigue instead. I figured out I can live on only protein (has some fat inevitably) and low carb vegetables, so I do. I can eat a lot if I keep my fats low. My treats are baby carrots and pure (baking 100%) chocolate. I do this so my brain is very clear and the (genius?) part of my makeup can thrive. My genius is my subconscious. I come up with wild ideas which often fizzle in the light of day but when they don't it is amazing. .Hi
I feel you are thinking of a keytone meter...?
Keytones are present when BGs go dangerously high.. Ketoacitosis. Not to be confused with ketones from fasting or a keto style diet with a normal safe BG level..
We use our BG meters (& or CGMs.) to detect ot preempt any blood sugar issues accordingly, correcting with the only medication to do the job. Exogenous "insulin" if we go too high. Basically, & on the whole. Anything we swallow (baring water, off the top of my head.) will have either a nominal immediate effect or big impact on BGs, or possibly latter on. Depending on the complexity of the carbs & the amount, or fats slowing the digestive process ultimately converting to blood sugar to power the body.. Which is why the correct insulin to carb ratio is imperative. Along with the timing of dose to compliment the carbs. Certain insulins have a certain working profile..
Unless our BG goes lower than healthy perameters. Then we use carbs..
Hey I'm just a "layman."
All I know is my beta cells took a beating during an autoimmune episode 42 years ago.
& personally, I feel yer barking up the wrong tree? Sorry.
Hope this helps!
HiI do not understand why anyone would want to simulate 'severe Type 2 Diabetes'. Isn't it wiser to try to ameliorate the effects of Type 2 Diabetes by changing one's lifestyle/medications etc?
If you are convinced that you have the 'hunter gatherer gene' then why not try to emulate a hunter gatherer type diet, one that is rich in sat fat and protein while low in carbohydrates.
I thought I answered that some people with reactive hypoglycemia eat diets which never lead to an insulin reaction because their blood sugar never gets high enough to trigger an insulin release. We are not all people with reactive hypoglycemia but we exist even if we end up anti social. If I ever had an insulin reaction to process sugar I would have major fatigue. I want my brain to be as clear as it recently has been and I therefore eat my strange diet.Hi
I feel you are thinking of a keytone meter...?
Keytones are present when BGs go dangerously high.. Ketoacitosis. Not to be confused with ketones from fasting or a keto style diet with a normal safe BG level..
We use our BG meters (& or CGMs.) to detect ot preempt any blood sugar issues accordingly, correcting with the only medication to do the job. Exogenous "insulin" if we go too high. Basically, & on the whole. Anything we swallow (baring water, off the top of my head.) will have either a nominal immediate effect or big impact on BGs, or possibly latter on. Depending on the complexity of the carbs & the amount, or fats slowing the digestive process ultimately converting to blood sugar to power the body.. Which is why the correct insulin to carb ratio is imperative. Along with the timing of dose to compliment the carbs. Certain insulins have a certain working profile..
Unless our BG goes lower than healthy perameters. Then we use carbs..
Hey I'm just a "layman."
All I know is my beta cells took a beating during an autoimmune episode 42 years ago.
& personally, I feel yer barking up the wrong tree? Sorry.
Hope this helps!
Hi
I have the gene but I don't want to be fat. If I eat the amount of food a "normal" person does I get fat. I use much less energy in processing sugar then "normal" people do. A lot of Indians have my gene since I can survive famines much longer than you unfortunates. The women with my gene pay since they become diabetic in pregnancy and lose more babies (and sometimes themselves) then you "normal's". I explain that we use the same (very similar?) hormones as does the placenta to protect the fetus/baby and if the women stopped the hormones making us insulin resistant the baby/fetus would die.
HiCan you enlighten me please, what are the names of these hormones? And the names of these placental hormones?
BTW it is insulin that makes us insulin resistant through the failed mechanism of carb metabolism. It is a chicken and egg scenario but hyperinsulinaemia plus hyperglycaemia = metabolic dysfunction. This would be for T2, anyway.
Zero carb isn't zero protein so gluconeogenesis will continue to produce the glucose you need to function quite happily. This is what the OP seems to be misunderstanding.. All the carnivores are approximately zero carb (apart from a few incidentals).I'm confused by this discussion. I always thought that T1s need a small amount of basal insulin, even if fasting or eating a zero carb diet, because the liver will supply glycogen to keep the brain supplied with energy/glucose.
But, if you're on a zero carb diet, you stop producing glycogen and then rely on fats for energy (or so says Dr google). So where does the carbohydrate come from to keep blood glucose levels up in the normal 4-7 range? Or is no one (apart from Inuits) on a zero carb diet?
Explain your analysis. I assume that over 50 years ago when these events occurred, before I realized I had a low blood sugar problem, I got very low blood sugar.
We know from the study done by Drenick et. al. (ref 9) that in non-diabetic obese persons who fasted for 2 months and achieved blood beta-hydroxybutyrate (BHB) levels of 8 mM when given a single dose of insulin to induce severe hypoglycemia suffered no symptoms despite BG values as low as 9 mg/dl (0.5 mmol/l).
@kohongw Can I ask you to explain 'flatline glucose' please? I am curious because I did keto for a while although now have reverted to low carb for a while. What level would you say was flatline glucose? Do you mean that the level is constant all the time the person is in ketosis? Just trying to get my head round this.
Thanks, yeh that's mostly what my chart looks like normally anyhow, whether low carb or 'proper keto'. At the moment I am having just the one blip with my evening meal, but that's because I have relaxed things a little. Thanks for answering.That is actually a term used by Cyrus Khambatta in his debunking keto myths videio
But if you follow the TypeOneGrit group, you would often see CGM with relatively flatline...on a good day, I get something like this...it I stay off carbs. The morning blimp was just eggs and cheese.
View attachment 29670
And we pretty much have to keep it at that tight range if we want to go into ketosis...otherwise our background insulin will still be too high to produce adequate ketones. So the easiest way is really to just fast for 24-72 hours...
Millions of people around the world who eat a ketogenic diet, achieve a flatline blood glucose profile and greatly reduce or eliminate their need for oral medication and insulin.
I have heard of someone getting ketones of say 2.0 with BGs of around 10-12. Now I wouldn't have thought that was possible, but maybe it's because of certain drugs that they take? Surely they can't be fat adapted with BGs that high, and it can't be the beginnings of ketoacidosis with BGs that low! If you see what I mean. I need to have less than 25g daily for about 7-10 days to get to the 2.0 level.Here is Cyrus' attempt to debunk Keto...so the word is out on how to get flatline glucose...millions are doing it. Do we still have a T2D crisis?
https://www.masteringdiabetes.org/ketosis-ketogenic-diets-misleading/
I have heard of someone getting ketones of say 2.0 with BGs of around 10-12. Now I wouldn't have thought that was possible, but maybe it's because of certain drugs that they take? Surely they can't be fat adapted with BGs that high, and it can't be the beginnings of ketoacidosis with BGs that low! If you see what I mean. I need to have less than 25g daily for about 7-10 days to get to the 2.0 level.
Thank you, that explains a lotPerhaps they are on SGLT2 inhibitor. It is known to induce ketosis
This is one possible mechanism...
https://www.ncbi.nlm.nih.gov/pubmed/28303514
This paper goes into greater details.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4525004/
In any case, it is un-natural and likely dangerous to have ketones with high glucose level. That's why exogenous ketones should also be considered with care.
HiI'm confused by this discussion. I always thought that T1s need a small amount of basal insulin, even if fasting or eating a zero carb diet, because the liver will supply glycogen to keep the brain supplied with energy/glucose.
But, if you're on a zero carb diet, you stop producing glycogen and then rely on fats for energy (or so says Dr google). So where does the carbohydrate come from to keep blood glucose levels up in the normal 4-7 range? Or is no one (apart from Inuits) on a zero carb diet?
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