How they can class "severe insulin deficiency " as T2 is completely beyond me..
Do they not use simple logic any more?
I am a big fan of Lund Uni's sub-groups (MODs, MARDs, SIRDs, SIDDs and SAIDs) - had hoped that they would take off but have come to accept - they haven't.
. (And Swedes are nothing if not supremely logical! Imho.)
In the sub-grouping there is a clear distinction of those who were diagnosed (correctly one assumes! At least for Lund Uni's argument) between SAID - Severe Auto Immune Diabetes, which of course is the auto-immune form, including LADA, and SIDD, which is caused by pancreatic burn-out or environmental/pharmaceutical/surgical damage to the pancreas and therefore one's own insulin production., that you mention above.
The genetic component mentioned in a post above (what an interesting read it all is indeed), is accounted for in these sub-groupings, as the Swedes found these five types of diabetes to be genetically distinctive. This makes sense to me, as I often boil down 'genes' to what we can see in body types and ancestral differences which mean one's metabolism and/or tendencies play out in different ways, including of course - to evolutionary recent food sources (like wheat and dairy). I've read Jenny Ruhl again recently, and she talks about the multiple factors contributing to blood glucose system breakdown in a very accessible logical way. And her citations are brilliant.
The Lund Uni researchers also associated certain complications with certain sub-groups - of particular interest to me as a SIRD Kiwi, as kidney problems are hugely associated with SIRD - which I like to point out to medical professionals and associates when I fight for subsidised care, as kidney dialysis is very expensive for our healthcare systems. (A losing battle in Kiwiland, sadly, although a highly logical argument - I am seen as an outsider in the system rather than a flag bearer, but you know - many of us are and we are hoping for bigtime change.)
Retinopathy is associated with SIDD, for those interested in their findings.
I do have a wee rave about how this links up with remission-pathways, but I am trying to keep this post digestible
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