I had bad experiences with two statins.
Currently I have high cholesterol but good BG control, a sensible weight (12 stone, six foot tall), and an active lifestyle, and normal blood pressure. Scan for aortic aneurysm in the abdomen a year or so ago seemed to show no issues and a good lining to at least one major blood vessel.
So barring an in depth and intrusive investigation of my vascular system I see no obvious evidence that my high cholesterol is causing me any problems.
I do know people with familial hypercholesterolaemia who have had blocked blood vessels, needed stents, in one case a quadruple bypass. However in those cases the problems manifested some time ago and they are of similar ages. I don't THINK I have that problem.
"
AAAs have traditionally been considered to be a
manifestation of atherosclerosis (7). However, this
conventional theory has been increasingly challenged
in recent years. Although many AAAs are accompanied
by pathologic evidence of atherosclerosis, a
causal relation has not been confirmed. Since most
persons with atherosclerosis do not develop an AAA,
it is likely that even if atherosclerosis does play some
role in AAA pathogenesis, additional etiopathologic
processes are involved. This is supported by research
at the cellular level that has shown pathologic and biochemical
differences between atherosclerotic occlusive
and aneurysmal disease of the aorta (8-14). For
example, in comparison with aortic tissue in atherosclerotic
occlusive disease, aneurysmal aortic tissue is
characterized by a greater amount of proteolytic activity
(8-12) and inflammation (13, 14). The determinants
and implications of these findings remain
unclear. However, the familial aggregation of incidence
of AAAs suggests that genetic susceptibility
may play a key role in their pathogenesis (15, 16).
Furthermore, risk factors for AAA may be different
than those for atherosclerosis. Lilienfeld et al. (5) have
pointed to epidemiologic differences between coro-
nary heart disease and stroke and AAAs as evidence
supporting this hypothesis.
In previous analytic studies of risk factors for AAAs,
cigarette smoking was the only atherosclerotic risk
factor that has been consistently associated with AAA
(17-26). With respect to other known atherosclerotic
risk factors, such as hypertension and hypercholesterolemia,
results from previous studies have been
inconsistent (19, 21-24, 27-30). We conducted a casecontrol
study in Winnipeg, Manitoba, Canada, to
investigate the association between AAAs and atherosclerotic
and other risk factors for AAAs."
https://oup.silverchair-cdn.com/oup...n-nFkbvrOA__&Key-Pair-Id=APKAIUCZBIA4LVPAVW3Q