Diet

[sally and james]

"50% of Type 2 diabetics will be on insulin within 6 to 10 years of diagnosis", therefore, quite clearly, 50% WILL NOT be on insulin and I bet that the low-carbers will be in the "Nots".
Sally

 

[jack412]

@Ruth B and @nooboots I guess after we lose weight and get our insulin resistance down through diet and exercise, whether/when we move onto insulin will depend on, how many and how good are our beta cells.

they say at T2 diagnosis about half of our beta cells are wacked
http://care.diabetesjournals.org/content/34/Supplement_2/S258.full
There is a progressive deterioration in β-cell function in patients with type 2 diabetes. At diagnosis, islet function may be reduced by up to 50% compared with healthy control subjects, and there is also likely to be a reduction in β-cell mass of up to 60%. The reduction in β-cell mass is due to accelerated apoptosis. Currently, few pharmacological therapies address this reduction in β-cell mass and function. This means that patients are generally subjected to an increasing polypharmacy to control their diabetes, with most eventually being treated by insulin

 

[jack412]

"50% of Type 2 diabetics will be on insulin within 6 to 10 years of diagnosis", therefore, quite clearly, 50% WILL NOT be on insulin and I bet that the low-carbers will be in the "Nots".
Sally

being a LCHF'er, I hoping so, I hope I have enough beta cells left, to last till something major goes, like a heart failure or something when I'm a 100

 

[Ruth B]

I think our best hope, long term, has to be stem cell research and the possibility of re-growing new beta cells from injected stem cells. It's still a long way off, but, just maybe, some of us will be around to see it happen.

 

[Bluetit1802]

Surely to goodness much also depends on how early we are caught. An early diagnosis, and quickish good control from that point must be taken in to account, but is it in these surveys?

 

[sally and james]

Many people on here respect Jenny Rhul "Blood Sugar 101" website. You can find it here, http://www.phlaunt.com/diabetes/14046702.php. I hope this link will go straight through to the page titled, "Do People with Type 2 Always Deteriorate", if not, you can get to it from the home page. I'll quote the first paragraph,
"The single most dangerous idea you are likely to encounter as you begin your struggle to live a healthy life with diabetes is the belief that science has proven, beyond a doubt, that no matter what you do, your Type 2 Diabetes will get worse."
I'm inclined to believe her.
Sally

 

[jack412]

one of her cite for that was a Japanese study, yet when I looked at it. all of the people were on insulin, I find it difficult to see your point. If it's other than to do what it takes including insulin to get your BG within range.
which is something I happen to agree with

http://journal.diabetes.org/diabetescare/FullText/Supplements/DiabetesCare/Supplement400/B21.asp

RESEARCH DESIGN AND METHODS — A total of 110 patients with type 2 diabetes (55 with no retinopathy [the primary prevention cohort] and 55 with simple retinopathy [the secondary intervention cohort]) were randomly assigned to multiple insulin injection therapy (MIT) groups and administered three or more daily insulin injections or assigned to conventional insulin injection therapy (CIT) groups and administered one or two daily intermediate-acting insulin injections. Worsening of microvascular complications was regularly assessed during 8 years. Two or more steps up in the 19 stages of the modified Early Treatment of Diabetic Retinopathy Study classification in retinopathy and one or more stages up among three stages in nephropathy (normoalbuminuria, microalbuminuria, and albuminuria) were defined as worsening of complications.
RESULTS — In both primary prevention and secondary intervention cohorts, the cumulative percentages of worsening in retinopathy and nephropathy were significantly lower (P < 0.05) in the MIT group than in the CIT group. In neurological tests after 8 years, the MIT group showed significant improvement (P < 0.05) in the median nerve conduction velocities (motor and sensory nerves), whereas the CIT group showed significant deterioration (P < 0.05) in the nerve conduction velocities and vibration threshold. From this study, the glycemic threshold to prevent the onset and progression of diabetic microvascular complications was as follows: HbA1c <6.5%, fasting blood glucose concentration <110 mg/dl, and 2-h postprandial blood glucose concentration <180 mg/dl.
CONCLUSIONS — Intensive glycemic control can delay the onset and progression of the early stages of diabetic microvascular complications in Japanese patients with type 2 diabetes.

 

[nooboots]

@Ruth B and @nooboots I guess after we lose weight and get our insulin resistance down through diet and exercise, whether/when we move onto insulin will depend on, how many and how good are our beta cells.

they say at T2 diagnosis about half of our beta cells are wacked
http://care.diabetesjournals.org/content/34/Supplement_2/S258.full
There is a progressive deterioration in β-cell function in patients with type 2 diabetes. At diagnosis, islet function may be reduced by up to 50% compared with healthy control subjects, and there is also likely to be a reduction in β-cell mass of up to 60%. The reduction in β-cell mass is due to accelerated apoptosis. Currently, few pharmacological therapies address this reduction in β-cell mass and function. This means that patients are generally subjected to an increasing polypharmacy to control their diabetes, with most eventually being treated by insulin

Crikey that all sounds very technical. I wonder if my OH could get a test to see what degree of functioning he has? As I learn more about diabetes over the years, I think he must be very lucky. He was bought up without eating regularly, never had breakfast and usually ate one meal a day which was usually fatty chips. As a young adult he ate a lot of sweet things simply for energy, he doesnt really have a sweet tooth and used to drink lots of coke and things. Then he got diagnosed with T2 about 10 years ago and didnt really take it all that seriously but did stop with the overtly sweet foods. So to be able to bring his levels down to the degree he has, just by cutting out some starchy carbs, not even going 'low carb', he has been very lucky. We dont think he has any degree of neuropathy, although there was some confusion over this when the nurse in May told him that he had a touch of retinopathy from a test from 2 years ago, and then when we both went to the appointment and his new eye test was on the screen she said that it showed no problems. So I hope her second comment was accurate.