All drugs can do that to the kidneys and liver @Mbaker . i know that is why you are dead against meds which is the right decision for you. But we arent all the same, unfortunately.I do have a different opinion on this, as LCHF is not limiting, for me it is liberating. Yesterday my daughter made a super creamy panacotta using Xylitol, it was far too sweet for me after a small mouthful, this would be the case for mainstream varb
Now that I have educated myself about the food groups, mentally I cannot face regular off the shelf carbs, such as biscuits and bread. As I am trying to get a 6 pack again I know I would have to work even harder. Finally for me I am comfortably achieving 1 meal a day at least 3 times a
I was furious, my eGFR count went down on Metformin and immediately back up without it; for something that could be "end-of-life" full disclosure I think is vital. All I was told was that Metformin does not induce a Hypo.
Research is going on in leptin resistance which opens up research for more insulin resistance. Some leptin deficiencies are genetical and as I've posted on a thread recently 3000 known genes are related to diabetes.I think the name “Type2” does not help and the NHS should start to measure insulin levels at health checks and treat the condition of “Insulin Resistance” as we all know it is a big risk factor for strokes, high BG, heart issues, dementia etc.
If someone is overweight with the fat on the middle it is very likely the issue is “Insulin Resistance” and a low carb diet is the best option, but yet my GP says “low fat”! I tend to think that Metformin should be offered to everyone with Insulin Resistance even if their BG is normal.
I expect some people who are labels Type 2 are closer to being “half a Type1”, e.g they are producing a level of insulin that is lower than a “normal person”, for some reason other than fat clogging up the system.
Maybe DNA testing will let us be classified into more useful groups allow GPs to give the same meds to everyone in a grouping….
I am losing weight on insulin injections because am treating the IR not assuming everyone on insulin is eating the wrong things so they can cover it with insulin like they presume type1s can.I so agree. Why use Gliclazide to stimulate the pancreas to produce more insulin without knowing how much the pancreas is already producing? People are drowning in insulin and gaining weight if they continue to eat to their medication, which is what the NHS seems to want us to do. No wonder it is seen as progressive.
I think you are both right. With what I know now, I would immediately do the Newcastle Diet (using real food, no satchets), I would then maintain on LCHF with exercise. I think it depends where you are in your journey; as I had never dieted a low cal diet would most likely have got me to where I needed to be initially, killing myself with over exercising I think was a harder root.
This is where I feel I'm going to get the best of both worlds. Bariatric surgery for supported 600cals (unless crazy and stupid enough to blend marsbars and the likes) and low carb lifestyle.Not convinced as too many people go back to their old ways…… Hence I think controlled carbs (say under 100g) should be the first angle of attract getting people eating in a way they will be happy with for the rest of their life’s. Then and only then use intermittent fasting or Newcashle Diet.
Asking people to do something that lots of people fail at is a bad option, but even if people fail to get down to low carb levels they are very likely to have learned to avoid the worse foods.
Dr Bernstein seemed to think so. Oh and my specialist who looks after the north of england.Is insulin a better option than Gliclazide because at least it gives the pancreas a bit of a rest, and unlike Gliclazide, people on insulin seem to be empowered to reduce their dose when they start to get BG under control?
Are you refering to the mix currently with metformin and victoza?As soon as SGLT2 inhibitors (Farxiga) have been in use for long enough for any long term side effect to be know, I think they should replace Gliclazide on the NICE flow chart, maybe even alongside Metformin without waiting to see if the Metformin works on its own.
I also think it may be worth offering Metformin and Farxiga to anyone that commits to “low carb training” without first waiting to see if diet and exercise works. Offer a small reward to anyone that controls their BG well enough for come off them, rather than wasting 3 months before they are started.
I think self monitoring of BG can be such a big motivator that it should be standard, but how do we stop people just writing down the number, without thinking about it and taking action when needed? Just having the Low Carb Training being led by people who have proved it works would be a great ideal.
I think most people with Type2 could turn it round, provided it is not left for many years to damage their organs.
Of course your right bgs being good isn't a sign of IR reversal.But it is only reversed until someone goes back to their old ways of eating..... Even on basic LCHF most people can go back to having carbs often, just not all the time. The problem is when people eat to their meters so never burn off enough fat by being "just enough low carb to get a good reading".
PS, does anyone want to buy our old bread maker, the bread it makes is so nice that you will get Type2........
[QUOTE="Daibell, post: 1503326, member: 21149"
Don't blame the GP's. What you describe here is exactly what the diagnosis guidelines laid down by NICE say, and follows their treatment Flow Chart exactly. By following this recipe exactly the GP is protected from any malpractice litigation,
But the point is that some have to use insulin treatment to stop high bgs damaging the body until the patient can be strong enough to fight back to improve their care. Most people who end up on insulin due to IR only are very ill/filled with circulating glucose which their own insulin isn't converting. That is why you get reviews on insulin therapy too. Things get worse and better.The key point is that Type 2 is caused by too much insulin in the body, so taking drugs to increase insulin or by injecting insulin you are making things worse, not better. Since 90% of type 2 diabetics are either overweight or obese, any increase in insulin which causes further weight gain can result in a higher risk of long term complications and shorter life expectancy.
Are you refering to the mix currently with metformin and victoza?
Of course your right bgs being good isn't a sign of IR reversal.
Maybe we need a new kind of meter?
This is where I feel I'm going to get the best of both worlds. Bariatric surgery for supported 600cals (unless crazy and stupid enough to blend marsbars and the likes) and low carb lifestyle.
I'm doing half already and have done 600cals in the past for 8-9mths losing 7stones. Oh and of course the IR/diabetes I wasn't aware of.
I won't be cocky enough to say I-'ll do it but I will have a very good shot at it!
Actually in the early days of T2D, the insulin resistance stops glucose being burnt off in the muscle tissues, and it congregates in the bloodstream, leading to possible DKA. The insulin resistance ALSO puts up barriers to the glucose being stored in the same cells (for future use) so actually one symptom of T2D is weight loss, not gain. So yes, one way of treating T2D is to flood the body with insulin to force glucose past the IR barriers so it becomes usable and storable again. Whilst this is a bad policy long term, it does prevent the deadly DKA, so saves lives. Ultimately the pancreas goes into burnout, and then insulin becomes totally necessary. The weight that glucose 'puts on' due to storage is a mixture of glucogen + water, and so when we cut carbs and glucose by diet then we lose weight due mainly to water loss. This plateaus out but is not the real cause of metabolic syndrome, which is actually caused by storing lipids in the liver and adipose cells.The key point is that Type 2 is caused by too much insulin in the body, so taking drugs to increase insulin or by injecting insulin you are making things worse, not better. Since 90% of type 2 diabetics are either overweight or obese, any increase in insulin which causes further weight gain can result in a higher risk of long term complications and shorter life expectancy.
But if you maintain healthy blood levels why take them ? I've recent seen my hda1c come down to 59 from 102 in 8weeks. I have stopped my Metformin and seen my bg level drop again.An interesting article.
So, Fung agrees with my decision that staying on Metformin is actually beneficial, and rushing to be med free may not actually be the diabetic's holy grail?
Metformin has other benefits it doesn't really reduce BG by much. My A1C has been 40 for the last year. And I will continue to take it.But if you maintain healthy blood levels why take them ? I've recent seen my hda1c come down to 59 from 102 in 8weeks. I have stopped my Metformin and seen my bg level drop again.
Should I consider my self lucky that I have been pescribed metformin ?
Metformin has other benefits it doesn't really reduce BG by much. My A1C has been 40 for the last year. And I will continue to take it.
For me it has been key in reducing PCOS symptoms.
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