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<blockquote data-quote="HSSS" data-source="post: 2382592" data-attributes="member: 480869"><p>I can only go on what I’ve read, as it’s not my experience, but many seem to think giving the beta cells a kicking in this situation is counterproductive, whereas preserving them with low carb/early insulin beneficial to prolonging the honeymoon. Like many things there are differing opinions and, on this, I don’t have one. </p><p></p><p>I personally don’t see LADA as a misdiagnosed type 1, more a slow onset, adult, version of type 1 that occurs within a few years of diagnosis. Perhaps others see it differently or I’m wrong. </p><p></p><p>Type 2 can still lose insulin supply over prolonged time without becoming LADA afaik. Though I can see how the two become blurred in the mid term, but I assume the type 2 retains an insulin resistance most type 1/LADA don’t have. I guess the difference can become somewhat moot as to the cause of loss of pancreatic function (assuming it’s permanent) given the treatment is much the same - insulin.</p></blockquote><p></p>
[QUOTE="HSSS, post: 2382592, member: 480869"] I can only go on what I’ve read, as it’s not my experience, but many seem to think giving the beta cells a kicking in this situation is counterproductive, whereas preserving them with low carb/early insulin beneficial to prolonging the honeymoon. Like many things there are differing opinions and, on this, I don’t have one. I personally don’t see LADA as a misdiagnosed type 1, more a slow onset, adult, version of type 1 that occurs within a few years of diagnosis. Perhaps others see it differently or I’m wrong. Type 2 can still lose insulin supply over prolonged time without becoming LADA afaik. Though I can see how the two become blurred in the mid term, but I assume the type 2 retains an insulin resistance most type 1/LADA don’t have. I guess the difference can become somewhat moot as to the cause of loss of pancreatic function (assuming it’s permanent) given the treatment is much the same - insulin. [/QUOTE]
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